Normal blood uric acid but still gout attack may be caused by catching a cold, long-term use of uric acid-lowering drugs and joint injury. 1. Cold: When the temperature drops, the solubility of uric acid decreases, so uric acid will precipitate in the form of crystallized salts and be deposited in various parts of the body. Once deposited in the joints or cartilage, gout may be induced. 2. Long-term use of uric acid-lowering drugs: Gout patients need to take long-term uric acid-lowering drugs, such as benzbromarone, in order to reduce the blood uric acid value to a reasonable range. If the patient’s blood uric acid level is well controlled, it will promote the dissolution of deposited uric acid, release these excess uric acid into the blood, and cause the blood uric acid level to rise rapidly, inducing gout. 3. Joint damage: Strenuous exercise, excessive walking and other conditions can lead to chronic damage to certain joints and an increase in the level of white blood cells in the joint fluid. Uric acid stimulates these white blood cells and induces gout. When a patient has a gouty attack, it is recommended to go to the rheumatology and immunology department of a regular hospital to find out the cause and follow the doctor’s instructions for medication.