Facial neuritis, facial palsy in Chinese medicine, is also known as facial nerve palsy, and its exact etiology is not fully understood. The stimulating factors may be wind and cold, viral infection and autonomic instability. The facial nerve travels in a narrow bony facial nerve canal, and once ischemic edema occurs, it inevitably leads to compression of the facial nerve. It can occur in both men and women, young and old. Facial palsy can occur throughout the year, but it is more frequent in winter and spring, i.e., the cold and windy seasons. Since the beginning of winter, there has been a significant increase in outpatient visits for facial neuritis. Facial neuritis usually starts acutely, with sudden paralysis of one side of the facial expression muscles, which can reach a peak within a few hours. Some patients have pain in the postauricular mastoid area of the affected external auditory canal 1-3 days prior to the disease, often found in the early morning when washing or by others. On examination, the ipsilateral forehead lines disappear, the eyebrows cannot be frowned upon, and the eyelid cannot be closed or is incompletely closed when the eye is closed due to paralysis of the orbicularis oculi. The lower eyelid is ectropioned and the tears do not flow easily into the nasolacrimal duct and spill out of the eye. The nasolabial folds on the diseased side become shallow, the corners of the mouth droop, and the corners of the mouth are drawn toward the healthy side when showing the teeth. The corner of the mouth is drawn toward the healthy side when the teeth are shown. The mouth cannot be puckered or whistled, and the cheeks are puffed into the corner of the mouth on the diseased side to leak air. Due to the paralysis of the buccal muscle, food is often trapped between the teeth and cheeks. In a few cases, there may be ipsilateral loss of taste sensation in the first 2/3 of the tongue, ipsilateral salivary and lacrimal gland secretion disorders, pain in the ear and behind the ear, and herpes zoster in the external ear canal and auricular area. Do not be nervous when you find the above symptoms. Facial neuritis is different from cerebrovascular disease in that there are no symptoms such as limb paralysis. Go to the hospital as early as possible when symptoms appear. In the early stage, the main treatment is to improve local blood circulation and eliminate inflammation and edema of the facial nerve, and in the later stage, the main treatment principle is to promote the recovery of nerve function. Pharmacological treatment: corticosteroid, oral prednisone 30mg/d, gradually reduce the dosage in 7-10 days after 5 days; B vitamin therapy, vitamin B1 100mg, vitamin B12 500µg intramuscular injection, in addition to the application of some microcirculation and vasodilator drugs, such as herpes virus infection, additional antiviral drugs; physical therapy: stem mammary foramen Nearby ultrashort-wave heat therapy, infrared radiation, and acupuncture during the recovery period are possible. In addition, to protect the exposed cornea to prevent the occurrence of conjunctivitis and keratitis, eye shields, eye drops, eye ointment and other methods can be used. Facial neuritis generally has a good prognosis, and recovery usually begins 1 to 2 weeks after the onset of the disease and heals within 2 to 3 months. About 85% of cases recover completely without sequelae. However, those who have not recovered for more than 6 months have a poor prognosis, and some may have facial muscle spasms or facial muscle twitches. The prevention of facial neuritis should enhance physical fitness, pay attention to the warmth of the face and behind the ears during the cold season, avoid sitting or sleeping with the head facing the window gap, and avoid air conditioning or blowing fans in summer to prevent the onset or recurrence.