The ideal treatment for HFS should eliminate spasticity symptoms while preserving the function of the facial nerve intact. With the development of manifestation surgery technology, the use of manifestation microvascular decompression (MVD) for the treatment of HFS is the most effective treatment method for HFS at present because of its small trauma, high cure rate, low incidence of surgical complications, and especially its ability to completely preserve the nerve and vascular function. The vascular compression of the facial nerve exit bridge zone (REZ) is considered to be a common cause of HFS. However, the definition of REZ varies among authors. Traditionally, REZ is considered to be the junction between the central and peripheral segments of the facial nerve, also known as the Obersteiner-Redlich zone. In fact, clinically, it mostly refers to the central segment of the cranial nerve, which is very sensitive to vascular compression, while the peripheral segment, which is wrapped by Chewang cells, is more resistant to vascular compression, so the responsible vessels are mostly found in the central segment. 2. Occupational lesions Occupational lesions are also a cause of HFS formation, also known as “secondary facial spasm”. Tumors in the pontocerebellar angle (CPA) have been reported in the literature to cause facial spasm in about 0.8% of cases. These tumors can be meningiomas, epidermoid tumors, or nerve sheath tumors, and can be caused by tumor compression alone or by simultaneous compression of tumors and blood vessels. Although the exact cause of Bell’s facial palsy remains unclear, it is now widely believed to be caused by inflammation, which leads to vascular compression of the facial nerve as well as local ischemia and demyelination, resulting in the development of HFS. In addition, Hideto et al. reported five cases of familial HFS and found no significant difference in clinical manifestations between familial and decentralized, and there was no exact inheritance pattern, possibly due to low episomal autosomal. Second, treatment 1, drug treatment hemifacial spasm usually requires surgical treatment. Early and less severe cases can be treated internally. Carbamazepine and phenytoin are usually ineffective in treatment, which is different from the case of trigeminal neuralgia with a clear etiology. Local injections of botulinum toxin (Oculi-num) may be effective in the treatment of hemifacial spasm and/or blepharospasm. 2. Surgical treatment Many excisional treatments are effective for hemifacial spasm (including severing facial nerve branches), but can leave patients with varying degrees of facial paralysis. The current treatment for HFS is mainly MVD, in which the blood vessel infringing the nerve is removed from the nerve and a sponge (e.g. Ivalon polyethylene formyl alcohol foam) is placed between the two as a padded isolation. The results of other pads are less satisfactory in comparison (the muscle may disappear and the Teflon pad may be thinner. Teflon pads are currently the most widely used padding material in China.