Hepatic ascites is mainly caused by portal hypertension caused by cirrhosis, and the mechanism of its formation is related to a variety of factors, such as decreased plasma colloid osmotic pressure, increased hydrostatic pressure, obstruction of lymphatic return, and diminished antidiuretic hormone inactivation. Clinically, cirrhosis can also be divided into compensated and decompensated stages according to the presence or absence of ascites. At present, ascites is divided into four main stages: first, pre- ascites, when the patient has not yet developed liver ascites and is located in the compensated phase of cirrhosis. Second, the reactive ascites stage, at this time, after appropriate application of diuretics, or the joint application of human albumin, can better control the symptoms of ascites. Third, the stage of intractable ascites, when the amount of ascites is more, clinical treatment is very difficult, sometimes only through TIPS or diversion surgery to reduce ascites. Fourth, the liver and kidney syndrome period, when the blood perfusion of the kidney is reduced, clinical symptoms of oliguria and anuria will appear.