Hyperthyroidism is a thyrotoxicosis caused by excessive production of thyroid hormones by the thyroid glands themselves. The effects of hyperthyroidism during pregnancy are unique, and the main effects on the fetus are: 1. The maternal thyroid hormone receptor stimulating antibodies (TSAb) can stimulate the fetal thyroid gland through the placenta, causing fetal or neonatal hyperthyroidism. 2. Hyperthyroidism can cause long-term maternal sympathetic excitation, increased oxygen consumption, decreased ATP stores, and insufficient fetal energy supply, resulting in fetal hypoxia and growth restriction, thus increasing the incidence of low birth weight and preterm infants, and increasing the mortality rate of newborns and perinatal infants. The negative effects of hyperthyroidism on pregnancy include miscarriage, premature birth, pre-eclampsia and placental abruption. Pregnancy is also a relative contraindication to surgical treatment, because surgery can cause premature birth and miscarriage. Therefore, in order to avoid the above, if the patient’s hyperthyroidism is not controlled, it is recommended not to get pregnant; if the patient plans to get pregnant, she can get pregnant when the anti-thyroid drug treatment, serum TT₃ and TT₄ reach the normal range, stop the anti-thyroid drug or apply the minimum dose of the anti-thyroid drug; if the patient is found to have hyperthyroidism during pregnancy and chooses to continue the pregnancy, in principle Undergo antithyroid medication and surgical treatment in mid-pregnancy.