Many sufferers go directly to some pharmacies to purchase drugs for treatment after discovering their condition, and without understanding their condition, use improper means of treatment to delay their condition. 1, drug treatment: facial spasm treatment commonly used drugs include carbamazepine, oxcarbazepine and valium. The maximum dose of carbamazepine for adults should not exceed 1200mg/d. Alternative drugs are phenytoin sodium, clonidine, baclofen, topiramate, gabapentin and haloperidol. Medication can reduce the symptoms of facial muscle twitching in some patients. Medication for facial muscle spasm is often used in the early stage of the disease, in those who cannot tolerate surgery or refuse surgery, and as an adjunctive treatment for those whose symptoms cannot be relieved after surgery. It should be noted that the sensitivity of facial spasm and trigeminal neuralgia to drugs such as carbamazepine is still different, and the remission rate of facial spasm is not high with simple application of drugs such as carbamazepine, and there may be adverse effects such as liver and kidney function impairment, dizziness, drowsiness, leukopenia, ataxia and tremor with drug treatment. Adverse drug reactions occur immediately discontinue the drug. In particular, there is a risk of exfoliative dermatitis with carbamazepine treatment, and severe exfoliative dermatitis can be life-threatening. 2, acupuncture: minimal trauma, but not curative, and too many acupuncture puncture operations are likely to induce facial nerve endings scarring and adhesions, prone to collateral movement. 3.Local closure: Generally, local injection of hormone or local anesthetic drugs near the stem mammary foramen on the same side of the ear causes a transient decrease in facial nerve function, replacing spasticity symptoms with mild facial palsy symptoms. Generally, as the symptoms of facial palsy are reduced, the spasm is gradually aggravated and returned to the pre-treatment level. Botulinum toxin type A for injection (botulinumtoxinA): It is mainly used in adult patients who cannot tolerate surgery, refuse surgery, fail surgery or relapse after surgery, or have ineffective medication or drug allergy. 90% of patients are effective for the first injection of botulinum toxin, and the time for complete relief and significant improvement of spasticity after one injection is 1~8 months, mostly concentrated in 3~4 months. The efficacy of Botox gradually decreases as the duration of the disease increases and the number of injections increases. The interval between two treatments should not be less than 3 months. If the treatment fails or the efficacy gradually decreases after repeated injections, other treatment methods should be considered. Therefore, Botox injections cannot be used as a long-term treatment for facial muscle spasm. It should be noted that the effect after each injection is closely related to factors such as the choice of injection site, the size of the injection dose and whether the injection technique is skilled. Adverse reactions to Botox injections include transient symptomatic dry eye, exposure keratitis, lacrimation, photophobia, diplopia, ptosis, reduced transient eyes, incomplete lid closure, and varying degrees of facial palsy, which may recover spontaneously within 3 to 8 weeks. Patients with repeated botulinum toxin injections will experience permanent eyelid weakness, shallow nasolabial folds, crooked corners of the mouth, facial stiffness, and other signs. Microvascular decompression surgery (MVD): In the 1970s, with the clinical application of the operating microscope, Jannetta took the lead in perfecting and standardizing the theory and surgical operation techniques of microvascular decompression, and popularized and popularized the surgical technique internationally. (1) Principle of surgery: The purpose of facial spasm surgery is to completely separate the responsible blood vessels compressing the facial nerve root from the facial nerve, and the full name of the surgery is: facial nerve root microvascular decompression, or MVD surgery. During the surgery, the adhesions around the nerve and blood vessels are fully separated, and non-absorbable Teflon cotton is padded between the blood vessels and the brain to achieve the effect of releasing the nerve compression. The facial nerve root is exposed under a microscope through the natural gap between the cerebellum and the skull, the compressing blood vessel is found, the adhesions between the vessel and the nerve are separated, and the compressing blood vessel is pushed away from the facial nerve root through Teflon cotton of a certain size (about the size of a green bean). How to treat facial spasm medication to be more effective, must be under the guidance of a doctor for medication, if the condition is serious also need to be surgical treatment.