End-plateau osteochondritis is often associated with lumbar disc degeneration and is one of the causes of lower back pain. Vertebral end-plateosteochondritis, referred to as end-plateosteochondritis, is an aseptic inflammatory disease of the cartilage. It is easily missed and misdiagnosed clinically. Diagnostic imaging is also underappreciated, and x-ray and CT have limited ability to visualize cartilage. Early imaging diagnosis of vertebral endplate in lumbar disc degeneration can help clinicians choose a timely and beneficial treatment pathway. The cartilage endplate is histologically thin, hyaline cartilage, and during development, it is the epiphyseal nucleus below the upper vertebral body, which is closely associated with the growth of vertebral body height. It is a flat disc-shaped structure with a thicker circumference and thinner center, and the anterior edge is thinner than the posterior edge, and the thickness is basically the same on both sides. Its role is to withstand pressure and protect the vertebral body, and as long as the cartilage endplate remains intact, the vertebral body is generally free of bone resorption due to pressure. The cartilage endplate is a multi-component system that prevents nucleus pulposus from protruding into the vertebral body, controls nutrient infiltration of the disc, and carries a cushion load, and is closely associated with disc degeneration by sharing spinal motion and bearing functions with the disc and maintaining normal disc morphology and physiological function. bone. Morphologic changes include signal abnormalities, thinning, fracturing, and loss of bone. There are also quantitative and qualitative changes in the biochemical composition of the subchondral bone tissue, such as metabolic product stagnation and fibrosis. Modic changes in the lumbar spine: Degenerative changes in the vertebral endplates and subchondral bone on MRI were first noted by de Roos et al. in 1987. In 1988, Modic et al. defined Modic changes for the first time in a study of 474 patients with chronic low back pain and classified them into three types: T1-weighted images showed low signal in the endplate and subchondral bone, while T2-weighted images showed high signal, with histological signs of edema and associated with increased vascularization of the endplate fracture and subchondral bone marrow. The histology shows edema and is associated with increased bone marrow vascularization in the subchondral bone and fissures, and a combination of microfracture, named modic type I degeneration. This endplate degeneration usually runs parallel to the edge of the disc and may extend 2 to 10 mm into the vertebral body, sometimes involving up to 50% of the vertebral body. In the sagittal view, it usually extends anteriorly to posteriorly, but rarely involves only the anterior or posterior portion of the vertebral body. On T1-weighted and T2-weighted images, it appears as a low signal, and histologically it is a modic type III degeneration with endplate and subendplate sclerotic bone. The distribution of modic degeneration in age and lumbar segments was observed, and it was found that the degeneration was mostly concentrated in the age group of 40-80 years, with the highest incidence in L4-5 and L5-S1.