Cirrhosis is a gradual process that can be divided into two stages: the compensated stage and the decompensated stage, each of which has its own manifestations. In compensated cirrhosis, which is often referred to as early cirrhosis, there are usually no obvious symptoms, and sometimes drowsiness and fatigue, loss of appetite and indigestion, or even diarrhea can occur, but they are generally not characteristic. This can generally be relieved by rest or other adjustments, which can easily lead to missed and misdiagnosed cases and eventually develop into the decompensated stage. Symptoms of the decompensated stage of cirrhosis include both hepatic decompensation and portal hypertension. Decompensated liver function is manifested by indigestion, aversion to greasy food, malnutrition, and in severe cases, jaundice begins to appear. In addition, since coagulation factors are synthesized in the liver, due to liver dysfunction, coagulation factor synthesis is reduced and, together with hypersplenism, patients develop bleeding and anemic symptoms. The activation and inactivation of hormones also need to pass through the liver, so the body will experience a series of endocrine disorder symptoms, such as abnormal sex hormones, irregular menstruation and infertility in women, and decreased libido, hair loss, breast development, spider nevus, and liver palms in men. The manifestations of portal hypertension are usually detected by examination, and are commonly found in the formation of portal vein collateral circulation, esophagogastric fundic varices, abdominal wall varices, hemorrhoidal varices, retroperitoneal anastomotic branch varices, splenorenal shunts, etc. Chronic portal hypertension also leads to hypersplenism, splenomegaly, and in more severe cases, peritoneal effusion. In severe cases, the signs are obvious and can be detected by the naked eye. End-stage cirrhosis, often referred to as advanced cirrhosis, often leads to a range of complications. Examples include vomiting blood and tarry stools, which are usually caused by bleeding from ruptured esophagogastric fundic varices. In addition, fluid accumulation in the abdominal cavity can lead to peritonitis, which in turn can lead to secondary abdominal infections. In more severe cases, it can lead to hepatic encephalopathy due to the accumulation of toxic substances in the blood, such as ammonia. In addition, it can also cause portal vein thrombosis, hepatorenal syndrome hepatopulmonary syndrome, and cancer. Therefore, the symptoms of cirrhosis are not obvious in the early stage, and the symptoms will be obvious only in the decompensated stage, so you cannot judge the presence of cirrhosis by symptoms alone, but also need timely examination for early detection.