Sympathetic cervical spondylosis has a wide range of symptoms, most of which are sympathetic excitation symptoms and a few are sympathetic inhibition symptoms. Since the surface of the vertebral artery is rich in sympathetic nerve fibers, when sympathetic nerve function is disturbed, the vertebral artery is often involved, resulting in abnormal diastolic function of the vertebral artery. Therefore, sympathetic cervical spondylosis is often associated with inadequate blood supply to the vertebral basilar artery system in addition to symptoms of several systems in the body. Clinical manifestations: Vertigo: Vertigo is a common symptom in patients with vertebral artery cervical spondylosis. Patients change position due to neck extension or rotation to induce vertigo symptoms. The vertigo caused by ischemic lesions of the vestibular nerve nucleus usually lasts for a short period of time and disappears in a few seconds to a few minutes, and the patient may have mild disorientation and movement disorders at the onset, manifesting as unstable walking or tilting to one side; vertigo caused by ischemic lesions of the vestibular nerve nucleus is not accompanied by impaired consciousness. The vertigo caused by vestibular neuropathy is central vertigo; the vagal ischemic lesion is peripheral vertigo. Some patients have nausea and cannot raise their heads during acute attacks. A few patients have diplopia, eye tremor, tinnitus and deafness. Headache: In patients with vertebral artery type cervical spondylosis, headache and vertigo symptoms usually coexist at the onset. Occipital neuropathy is the main cause of headache. Because the occipital artery, a branch of the vertebral artery, supplies the occipital nerve, clinically the vertebral artery causes ischemia of the occipital nerve and headache symptoms appear in the occipital nerve innervation area, which is intermittent throbbing pain, radiating from the back of one side of the neck to the occipital area and half of the head, with a burning sensation, and a few patients have nociceptive hypersensitivity, and the pain is obvious when touching the head. In addition, the rhomboid muscle, which is innervated around the paraspinal nerve, can cause spasm of the rhomboid muscle after root lesion or trauma to this muscle, and the occipital nerve branch that penetrates from the rhomboid muscle is squeezed to induce clinical symptoms. Displacement of the atlantoaxial or pivotal vertebrae can also stimulate the occipital nerve that penetrates through it and induce headaches. Visual impairment: Due to spasm of the vertebrobasilar artery system caused by cervical spondylosis, secondary ischemic lesions of the visual center of the occipital lobe of the brain may occur in a small number of patients with reduced visual acuity or visual field defects, or even blindness in severe cases.