Spinal cord embolism syndrome generally refers to a series of spinal cord and cauda equina dysfunctions caused by spinal cord tumors or simple shortening of the spinal cord end filaments due to multiple causes such as spina bifida (spina bifida, spinal cord spondylolisthesis, etc.), such as abnormal lower extremity development, abnormal sensory activity, and abnormal bowel movements (neurogenic bladder). MRI reveals morphological abnormalities and cone downward displacement of the spinal cord due to strain and compression. This is important in the diagnosis of spinal cord embolism, which is generally not diagnosed in the absence of morphological and positional changes in the spinal cord. Treatment is primarily surgical and should involve removal of the primary focus or (and) severance of the spinal cord end filaments to release adhesions and compression of the spinal cord and nerves. In general, surgery should be performed as soon as possible after the discovery of the disease, and the effectiveness of treatment depends on the specific condition. For most patients, treatment is effective in relieving symptoms and delaying the onset of symptoms to some extent, but surgery – even multiple surgeries – is not likely to completely resolve the spinal cord embolism, and the spinal cord is usually still visible on MRI after surgery. In fact, the spinal cord embolism can remain with the patient for life. If the lower extremity development or mobility is still impaired after surgery, orthopedic treatment should be provided, and if there is abnormal urination, treatment should be continued in urology and intermittent clean catheterization should be performed if necessary. In addition, injection of neurotrophic drugs and acupuncture treatment can be helpful in relieving symptoms.