Patients with renal tubular acidosis generally present with changes in blood potassium, such as hypokalemia, but patients with hyperkalemic renal tubular acidosis have elevated blood potassium. In addition, patients in this group may have abnormal biochemical indicators such as phosphorus and calcium, and patients with a type of Fanconi syndrome, where there is a multifunctional defect in the proximal renal tubules, may have abnormal blood biochemical indicators for substances such as amino acids, proteins and glucose.