Myocardial infarction is an occlusion of a coronary artery that interrupts blood flow, causing local necrosis of part of the myocardium due to severe and persistent ischemia. Clinically, there is severe and persistent retrosternal pain, fever, leukocytosis, accelerated erythrocyte sedimentation rate, increased serum myocardial enzyme activity and progressive electrocardiographic changes, and arrhythmia, shock or heart failure, or even sudden death may occur. Myocardial infarction is one of the major diseases that seriously endanger human health and is the main cause of death in patients with heart disease. Some data show that 2/3 of patients with acute myocardial infarction die before they are sent to the hospital. Therefore, it is important to shorten the time between the onset of the disease and arrival at the hospital, and to provide active treatment during this time to save the patient’s life. For patients with serious conditions, it is advisable to resuscitate them locally after the onset of the disease, actively contact doctors, and transfer them to hospitals for treatment only when their condition is stable and allows for transfer. How to detect acute myocardial infarction The most common people are those with a history of hypertension or hypertension before the onset of the disease, nearly half of the patients have angina pectoris, followed by smoking, obesity, diabetes and lack of physical activity are more likely to suffer from the disease; the onset of the disease is mostly in spring and winter, related to the cold climate and temperature changes; the onset of the disease is mostly without obvious triggers, often in quiet and sleep, and some patients develop the disease during strenuous physical labor, mental stress or after a full meal. Some patients have the onset of the disease during strenuous physical labor, mental stress or after a full meal, or even when straining to defecate. In addition, shock, bleeding and tachycardia can trigger the disease; about 20-60% of patients with acute myocardial infarction have aura symptoms; the most prominent symptom is pain, and its nature, attack time, accompanying sensation and sensitivity to nitroglycerin are very different from previous angina. Other symptoms include systemic symptoms such as fever, fatigue and sweating, gastrointestinal symptoms such as nausea, vomiting and epigastric distension, as well as arrhythmia, hypotension, shock and heart failure. The possibility of myocardial infarction should be considered when the above-mentioned symptoms occur in susceptible people, or in elderly patients with sudden onset of shock, severe arrhythmia, heart failure, epigastric distension or vomiting for unknown reasons, or in those with pre-existing hypertension and sudden decrease in blood pressure with no cause to be found, or in those with shock after surgery but bleeding and other causes are excluded. In addition, elderly patients with severe and prolonged chest tightness or chest pain should consider the possibility of this disease even if there are no characteristic changes in the electrocardiogram. All of them should be treated as acute myocardial infarction first, and electrocardiographic observation and serum enzyme measurement should be repeatedly performed within a short period of time to determine the diagnosis if available.