Chronic hypertrophic rhinitis is a hypertrophic change of the nasal mucosa, submucosa and turbinate bone, which generally develops from chronic simple rhinitis. The pathology of mucosal epithelial cilia is shed and becomes compound cuboidal epithelium, the submucosa is edematous followed by fibrous tissue hyperplasia and mucosal hypertrophy, for a long time, it can be mulberry-like or polyp-like changes, periosteum and bone tissue hyperplasia, the nasal turbinate bone can also be hypertrophic changes. Clinical manifestations 1, nasal congestion is heavy, mostly persistent, often open-mouth breathing, the sense of smell is mostly reduced. 2.The nasal discharge is thick, mostly mucus or mucopurulent. Due to the post-nasal flow, it stimulates the throat and causes cough and phlegm. 3.When the hypertrophied middle turbinate presses the nasal septum, it can cause the pressure or inflammation of the anterior sieve nerve from the ophthalmic branch of the trigeminal nerve, resulting in irregular episodes of frontal pain and radiation to the nasal bridge and orbit, called anterior sieve neuralgia, also called anterior sieve nerve syndrome. 4. Examination: ① The inferior turbinate is obviously enlarged, or both the inferior and middle turbinates are enlarged, which often leads to nasal blockage. There are mucous or mucopurulent secretions at the bottom of the nasal cavity or in the lower nasal passage. The mucosa is swollen, pink or purplish red, with an uneven surface or nodular or mulberry-shaped, especially at the front of the inferior turbinate and its free edge. The depression is not obvious when the probe is lightly pressed, and there is a hard and solid feeling when touched. Mucosal contraction is not obvious after using vasoconstrictor locally. Treatment 1. The application of vasoconstrictor nasal drops is limited to light cases. 2.Submucosal sclerosing agent injection of inferior turbinate, the mechanism of action is that after sclerosing agent injection, it can cause local chemical inflammatory reaction, produce scar tissue, reduce the volume of turbinate and improve ventilation. Commonly used 50% glucose solution with 15% sodium chloride solution, 5% sodium cod liver oil acid or 80% glycerin. After surface anesthesia of the turbinate, a 22-23 gauge needle is used to pierce the inferior inferior turbinate from the front end of the inferior turbinate parallel to the back, do not pierce through the mucosa, and inject sclerosing agent while withdrawing the needle until it is pulled out. It can also be injected at the front, middle and back of the inferior turbinate in 3 times, 0.5ml on each side, once every 10 days, with 3 to 5 times as a course of treatment. 3.Submucosal electrocoagulation of the mucous membrane of inferior turbinate to produce scar contraction. After surface anesthesia, the electric needle is stabbed from the front of the inferior turbinate, coagulated for 20-30 seconds and then pulled out, with a current of 10-30 mA. 4.Cryosurgery, a special freezing head is placed on the surface of the inferior turbinate to do freezing for 1 to 2 minutes each time to make the diseased mucosa necrotic and fall off and regenerate the mucosa. 5.Surgical treatment, general treatment is ineffective, or the mucosa is significantly thickened, or the thickened part is located in the posterior end or lower edge of the inferior turbinate, partial excision of inferior turbinate or partial excision of middle turbinate is feasible. In principle, it should not exceed 1/3 of the inferior turbinate to avoid affecting the function of nasal mucosa or secondary atrophic rhinitis. In the case of bony hypertrophy, submucous-periosteal resection of the inferior turbinate is appropriate, which can improve the ventilation and drainage of the nasal cavity without compromising the physiological function of the nasal mucosa. 6, for systemic chronic diseases or adjacent lesions such as deviated nasal septum or sinusitis, appropriate treatment is also given.