After vitreous hemorrhage, early bed rest should be taken in a semi-recumbent position so that the blood is deposited below so that the blood does not obscure the central vision, and force and strenuous head movements should be avoided. The vitreous itself has the ability of self-purification, and a moderate amount of vitreous hemorrhage can generally be absorbed by itself within 3-6 months. Whether drugs and ultrasound can accelerate the absorption of vitreous hemorrhage has not been confirmed conclusively. Hypertonic agents such as mannitol may promote the entry of blood components into the blood vessels and thus accelerate the absorption of vitreous hemorrhage. Chinese herbal treatment may be of some value. Early bleeding is given to cool the blood to stop bleeding, and after stabilization, blood is given to activate blood stasis and disperse nodules. Amintoiodine intramuscularly or Volitene (a drug containing organic iodine) orally may be helpful in the absorption of hemorrhage. However, it is important to note that during the conservative treatment period, the fundus must be checked regularly at the ophthalmology department, and when the fundus cannot be seen, an ocular ultrasound must be performed to understand whether retinal detachment or proliferative lesions occur, so that timely surgical treatment of vitreous hemorrhage is an effective measure. In general, surgery can be considered for those who have no significant effect after 3 months of conservative treatment. Vitreous hemorrhage caused by retinal vascular disease is mostly due to the rupture of retinal neovascularization, which can lead to repeated bleeding and then proliferative lesions, and the regression of neovascularization often requires retinal laser photocoagulation, while laser treatment is not possible when there is more blood accumulation in the vitreous, therefore, vitreous hemorrhage caused by this type of disease is currently considered to advance the timing of surgery to 1 to 2 months in order to avoid serious Proliferative lesions should occur, and the primary disease should be actively treated. Vitreous hemorrhage caused by ocular trauma, such as combined intraocular foreign body, endophthalmitis, etc., should be operated in a timely manner; if combined with ruptured eye injury, especially the thicker vitreous hemorrhage of scleral rupture injury but not yet retinal detachment, it is estimated that the possibility of self-absorption is small, and can be operated about 2 weeks after the injury to avoid the stimulation of blood to the eye tissue and reduce the chance of intraocular fibroproliferation, and 2 weeks after the injury when the vitreous body has mostly Posterior detachment occurs, and it is easy to surgically remove the vitreous blood without surgery-related complications. Vitreous hemorrhage caused by blunt contusion can be observed for 2 to 3 months if there is no significant absorption and no improvement in visual acuity. Anyone with combined retinal detachment should be operated early. In addition, vitreous hemorrhage (also called Terson syndrome) caused by intracranial hemorrhage for various reasons, because this kind of vitreous hemorrhage is mainly concentrated in the macular area of the posterior pole, not only has a great impact on vision, but also the destruction of macular tissue structure caused by the metabolic products of blood cells will lead to permanent damage of vision, therefore, if this kind of vitreous hemorrhage is observed for 1 month without obvious absorption, surgery should be considered Treatment. Vitrectomy can be combined with intraocular laser or extraocular condensation to perform photocoagulation or condensation on retinal avascular area due to retinal fissure and vascular disease, and close postoperative follow-up should be performed after surgery to supplement retinal photocoagulation if necessary to stabilize the condition, prevent rebleeding and protect useful vision.