The blood supply to the spinal cord is unique in that it is made up of small terminal arteries with thin lumens. As we age, the lining of the arteries gradually thickens and becomes brittle, and excess lipids in the bloodstream take advantage of this, making the already thin lumen of the arteries even narrower and reducing blood flow. As a result of these causes and congenital deficiencies, spinal cord ischemia occurs under specific triggers. An early manifestation of spinal cord ischemia is intermittent claudication. Since the activities of the lower extremities are governed by the spinal cord, when walking, the excitability of nerve cells in the spinal cord increases and the demand for oxygen and energy increases, and this is due to arteriosclerosis, narrowing of the lumen, and inadequate blood supply in the spinal cord, and this causes ischemia in the nerve cells of the spinal cord. After walking for a period of time, you will feel weakness in both lower limbs, and the more you walk, the more weak you feel, and finally you cannot walk. After a short rest, the weakness disappears because the blood supply to the spinal cord improves; however, the weakness returns when walking again, a phenomenon medically known as “intermittent claudication”, which is a yellow card warning of spinal cord ischemia. If effective treatment is carried out at this time, it is often possible to receive twice the result with half the effort. If the treatment is not timely and the ischemia continues to develop, spinal cord thrombosis may occur, resulting in bilateral lower limb paralysis, loss of sensation and urinary and fecal incontinence, causing lifelong regret.