First, the dynamic factors are mainly due to the loosening and dislocation of the hook vertebral joint after the instability of the vertebral joint, which then spreads to the lateral upper and lower transverse foramina, resulting in axial or lateral displacement, thus stimulating or compressing the vertebral artery causing spasm, stenosis or folding changes. This factor is the most common, mostly in the early light. Second, the vascular factor This factor is not only complex, and the vulnerability is large. The main manifestations are: 1, abnormal vascular dynamics. This disease is mostly seen after middle age, in addition to the degenerative factors of the cervical spine itself, it is also directly related to the weakening of the elastic retraction force of blood vessels. On the one hand, due to the role of age factors, on the other hand, with the high activity of the cervical spine, especially rotation, forward flexion, etc. are in the state of strain on the vertebral artery, thereby accelerating the degeneration of blood vessels. 2, arteriosclerotic changes. After middle age, the arteries of the whole body can appear sclerotic changes of different degrees, and the vertebral artery is no exception, the degree of which is proportional to age. If atheromatous plaques appear on the vessel wall (vertebral artery is one of the preferred sites), this process will be accelerated. 3. The effect of intervertebral space spacing changes on the vertebral artery. At the same time as the degeneration of the vertebral space, due to the narrowing of the spacing of the vertebral artery, the vertebral artery is relatively too long, prone to folding, thickening and bending changes, disrupting the original balance between the vertebral artery and the cervical bones, so that the blood flow is obstructed. 4, vascular variation. Anatomical material shows that the vertebral artery and vertebral vein (plexus) are prone to variation, including the separation of the transverse foramen (a few can be divided into two to three), the return of the vertebral artery sulcus above the cervical spine (bone ring formation), changes in sagittal and transverse diameter, differences in the number of vessels, asymmetry and caliber of the vessels on both sides, etc. are directly related to the occurrence and development of this disease. Several of these factors may occur simultaneously or one of them may predominate. The symptoms are easily eliminated by effective measures such as local braking if they are due to vertebral joint instability and local traumatic reactions. In contrast, those caused by mechanical factors such as proliferating bone spurs are mostly persistent. If several pathogenic factors coexist in the same case, when one of the reversible factors is eliminated after treatment and the symptoms disappear or are significantly reduced, it means that the other factors are not dominant and the prognosis is better. However, if the symptoms are not significantly relieved after various treatments, it indicates that mechanical compression is the main cause of the onset and development of the case, and surgical treatment is required on the basis of other disorders excluded. Zhang Jian et al [1] found that the distortion of the vertebral artery caused by hook joint hyperplasia and collapse of the vertebral space is the main cause of cervical vertigo. Through a comprehensive analysis of the latest research data on the non-osseous pathogenesis of vertebral artery cervical spondylosis in recent years, Wu Jinqiu et al. concluded that the main factors of its pathogenesis include soft tissue spasm and aseptic inflammation in the neck, elevated neuropeptides in body fluids, tortuous, thinning and sclerosis of the vertebral artery, and spasm of the vertebral artery caused by sympathetic nerve stimulation. Fan Dapeng et al [3] concluded that mechanical compression and sympathetic nerve stimulation are still the more recognized factors at present, and humoral factors and vascular lesions are also factors that cannot be ignored, and the vertebral artery dominance theory is a newer research result at present. Shi Da et al. believe that the back of the neck is where all the veins meet, and because of long-term head down, the pathway of qi and blood is closed, so that qi stagnation, blood stasis and phlegm coagulation in the collar and back form vertebral artery cervical spondylosis, so the pathomechanism of vertebral artery cervical spondylosis in middle-aged and young people is more actual and less deficiency, that is, phlegm and blood stasis blocking the ligament. Su Haitao et al. believe that the mechanism of vertebral artery cervical spondylosis in middle-aged and elderly people is mostly deficient in nature and real in symptoms, and the deficiency in nature is mostly due to deficiency of liver and kidney and deficiency of qi and blood, while the real symptoms are mostly due to phlegm and blood stasis blocking the ligaments.