Femoral head necrosis eating ammonia, i.e. glucosamine, is generally ineffective. Femoral head necrosis is a local bone trabecular fracture or femoral head cystic degeneration and collapse caused by impaired blood circulation in the femoral head, with pain and restricted movement of the affected hip joint as the main clinical manifestation. Glucosamine includes glucosamine hydrochloride, glucosamine sulfate, etc. The main components are glucosamine hydrochloride and glucosamine sulfate sodium chloride compound salt, which are mainly used to treat osteoarthritis in various parts of the body, such as knee joint, hip joint, and inflammation of wrist, spine, hand, shoulder joint and ankle joint. Femoral head necrosis is generally caused by trauma, heavy alcohol consumption, use of glucocorticoid drugs and other factors that lead to impaired and interrupted blood supply, bone marrow components, bone cell death and tissue repair, followed by structural changes and collapse of the femoral head, which eventually leads to osteonecrosis. In this case, you can use drugs that inhibit osteolysis and increase osteogenesis such as alendronate tablets and dobutamine tablets as prescribed by the doctor, or treat by surgical methods such as marrow core decompression and artificial hip replacement, while the application of glucosamine is usually ineffective. Once femoral head necrosis is detected, it is recommended to detect and treat it early, which can greatly improve the quality of life of patients and avoid the formation of generalized bone necrosis. When patients are not treated in time, or when the condition is poorly controlled with femoral head capsulitis and collapse, the symptoms of pain and limited limb movement will gradually increase, and when the joint movement is limited for a long time, it can be followed by lumbar spine osteoarthritis.