How does hydrocephalus occur secondary to cerebral cysticercosis?

  Cerebral cysticercosis is a disease of brain tissue damage caused by pig cysticercus parasiticus in the brain. It is common in northeast and north China, followed by northwest and Yunnan, and rare south of the Yangtze River. Tapeworm eggs that enter the stomach through the mouth hatch into cysticercus in the duodenum and enter the brain parenchyma, subarachnoid space and ventricular system by drilling into the intestinal wall and entering the body circulation and choroid through the intestinal membrane vein, causing various damages.  Intrinsic self-infection: In patients with tapeworm, due to vomiting or intestinal retrograde peristalsis, the tapeworm gestation segments flow back into the stomach, and the eggs hatch in the duodenum to form hexacercariae, which burrow through the intestinal wall into the small mesenteric veins and lymphatic circulation and are transported to the whole body and brain, where they develop into cysticercus larvae.  2, external self-infection: the tapeworm patient’s hands are contaminated with eggs, contaminated food, and infected through the mouth.  3.External infection: The patient does not have tapeworm parasites, but becomes infected after ingesting vegetables or fruits with eggs.  Ventricular cysticercosis can lead to blockage of cerebrospinal fluid circulation pathways, and cerebrospinal pool cysticercosis can cause subarachnoid adhesions, occlusion and arachnoid hyperplasia, affecting cerebrospinal fluid circulation and absorption, which can be secondary to hydrocephalus. In patients with cerebral cysticercosis, the cerebrospinal fluid contains a large number of inflammatory cells, with increased protein and even a cloudy appearance, so if a ventriculo-abdominal shunt is performed, the shunt is prone to blockage after surgery, resulting in surgical failure.  With neuroendoscopic surgery, the vesicles in the ventricles can be removed and the cerebrospinal fluid circulation pathway can be opened or reconstructed without placing a shunt, thus avoiding complications such as infection, blockage, and excessive drainage after shunt surgery.