Facial myoclonus is a muscle twitch on one side of the face (individuals present with bilateral spasms). It is characterized by paroxysmal involuntary twitching of the hemifacial muscles, usually limited to one side of the face and occasionally seen on both sides. The initial symptom of facial muscle spasm is eyelid fluttering, which usually does not attract attention, and gradually develops to the cheeks and the whole half of the face, and after a period of time it develops into a facial spasm, even to the corners of the mouth, and in severe cases, even to the neck. The more nervous and excited the person is, the more severe the spasm becomes. It can be aggravated by fatigue and tension, and in severe cases, it can be in spasm. In a few cases, mild facial paralysis may occur at the end of the process. Modern medicine believes that: a) The vast majority of cases are due to pathological disturbance of facial nerve conduction caused by some kind of compression, mostly due to crossed normal blood vessels compressing the facial nerve root, and occasionally due to compression by aneurysms or tumors. b) Another part of the cause is idiopathic facial nerve paresis with secondary facial muscle spasm on the affected side after recovery. This may be due to pathological changes in the demyelination of the nerve due to facial neuritis and failure to return to normal. c) In a very small number of patients, the spasticity of the affected facial muscles appears after trauma or surgery. Diagnosis and examination The diagnosis is mainly based on symptoms and signs, but cranial CT and MRI examination is needed to exclude other intracranial lesions. Treatment 1.Medication: Various sedative, tranquilizing and anti-epileptic drugs can reduce the symptoms in a few patients. 2, physical therapy: for some patients can reduce the symptoms. In recent years, radiofrequency therapy is used to temporarily interrupt the conduction function of the facial nerve with radiofrequency thermal coagulation to stop the seizures. 3.Ethanol injection therapy: injecting the facial nerve stem with different concentrations of ethanol can temporarily interrupt the conduction function of the facial nerve, so that the facial muscle twitching can be lifted. 4.Botulinum toxin injection therapy: Botulinum toxin is injected into the facial motor nerve terminals to produce muscle relaxation paralysis and achieve the purpose of relieving the spasm. 5.Triple injection therapy: according to the specific situation of the patient, hormone, Valium and other drugs are injected into the stem mammary foramen, the midpoint of the preauricular fold or the facial motor point, so as to relieve the facial muscle spasm. 6.Surgical treatment: At present, the most fundamental and effective treatment is facial nerve microvascular decompression. The procedure is to open the cranium behind the ear, expose the facial nerve, find the compressed blood vessel in the brain stem area, and pad it with gelatin sponge, muscle sheet or Teflon cotton sheet to achieve the purpose of decompression. The mortality rate of microvascular decompression is extremely low. Some patients may experience transient nausea, vomiting, or even hearing loss, facial sensory disturbance and vertigo after surgery. Only a very small number of patients may have permanent sequelae such as hearing loss, deafness, facial palsy, facial sensory disturbance, and brainstem infarction.