The typical symptoms often start with involuntary twitching at the corners of the eyes, with the onset of the lower eyelid being the most common, and can gradually progress to the corners of the mouth, and in severe cases can cause facial spasms and deformities. The frequency of these twitches is rapid and can be described as fine twitching. In severe cases, the twitching can progress to spasms, with the face resembling a cramp, eyes open, and the corners of the mouth tilted to one side. Facial muscle spasms are usually unilateral in onset. If the onset is bilateral, especially if the symptoms are closed eyes, eyes open, and strange movements of the eyes and mouth, it is often not facial myospasm. Treatment for facial myasthenia is currently available in the form of medications, facial injections with botulinum toxin, and microvascular decompression. Most of these medications are ineffective or only work for a period of time and then gradually fail. Local injection of Botox is only a symptomatic treatment, which cannot cut off the root cause, and the effect of one injection can only last 1-3 months in general. Since Botox is a neuromuscular paralyzing agent, it also causes local muscle paralysis, expression mina (local expression muscles are anesthetized), and also leads to a numb feeling. Since the location of the blood vessel compressing the facial nerve is located in the brain (deep in the ear in the brain, next to the brainstem, where the facial nerve emanates from the brainstem), microvascular decompression requires a craniotomy, but we currently use very small bone windows, ranging from about 2-3 cm, and the incision for the surgery is located behind the ear. The main operation of the surgery is to open a bone window (bone hole) in the skull, probe to depth, find the blood vessel compressing the The main operation involves opening a bone window (bone hole) in the skull, probing deeper, finding the blood vessels that are pressing on the facial nerve, and then separating the vessels from the nerve with a special material. The overall efficiency of the operation is about 97%, and if the operation is effective, only a very small number of patients may have a recurrence. The safety of the surgery is very high and we have not had any serious complications (complications that lead to death or disability of the patient) with our surgery so far. However, scientifically speaking as long as brain surgery is craniotomy, complications cannot be completely excluded. Among them, intraoperative or postoperative intracranial hemorrhage is the most dangerous complication that can be life-threatening, but the incidence is very low. Postoperative cerebrospinal fluid leakage, intracranial infection, facial palsy (about 6% incidence, mostly temporary), hearing loss, tinnitus, and facial numbness are most of the symptoms are temporary and recover completely or improve significantly with treatment and time. Pain from the incision, dizziness, nausea and vomiting may be present 1-3 days after surgery. Most patients can get out of bed 1-2 days after surgery and can be discharged 3-4 days after surgery if stitches are not removed in the hospital.