Facial muscle spasm is customarily called “facial twitching” and is characterized by repeated episodes of involuntary twitching of the facial muscles. Most of them are limited to one side, often occurring first in the eyelid, similar to eyelid jumping, and then gradually expanding in scope, mostly spreading to other facial muscles, the corners of the mouth, and even the broad neck muscles in sequence after several months. There is no aura before the attack, and the attack is characterized by rapid and frequent muscle twitching for a few seconds to a few minutes each time, and everything is as normal in the interval. Autonomous facial movements, chewing, transient eyes or random facial expressions can trigger facial muscle twitches, which can be aggravated by emotional excitement, stress, exertion or prolonged reading time, and the symptoms are reduced when resting or emotionally stable. If the seizure is serious, it can be twitching all day long, even during sleep. In some patients, the eyelid fissures may become smaller due to compulsive contraction of the eyelids, and in some patients, even the facial muscles may contract tensely, causing the corners of the mouth to be continuously tilted to the side of the disease. Medical experts have confirmed that facial muscle twitching is caused by compression of the intracranial segment of the facial nerve by abnormal blood vessels, resulting in damage to the facial nerve and demyelination of the facial nerve, resulting in a current “short circuit” between the nerve fibers and causing facial muscle twitching. In recent years, a large number of clinical practices have also shown that once the compression of the nerve by blood vessels is removed, facial muscle spasm can be stopped immediately or gradually. Since the cause of facial myospasm is intracranial, the only way to cure the disease is to treat the root of the disease.