Exertional angina is a type of angina that is characterized by angina induced by physical activity, emotional excitement, or other conditions that are sufficient to increase the oxygen demand of the myocardium, and the pain disappears rapidly after rest or sublingual nitroglycerin. The mechanism of angina pectoris is that although the coronary artery lesion causes narrowing of the blood vessel, the blood flowing through it at rest can still meet the myocardial oxygen demand, so there is no angina pectoris symptom, but when the heart activity is increased during physical activity or emotional excitement, more blood oxygen supply is needed, but the blood supply cannot be increased accordingly due to the narrowing of the coronary artery, which leads to myocardial ischemia and hypoxia and causes angina pectoris. Rest or medication relieves this contradiction between supply and demand and restores it to a state of equilibrium, so that the symptoms of chest pain are immediately relieved. Exertional angina is subdivided according to its clinical characteristics: primary angina, stable angina and worsening angina. Primary angina: It is the first occurrence of exertional angina for less than 1 month without previous angina or myocardial infarction. Patients who have had stable angina for several months without pain, now occurring again for less than 1 month can also be included in this type. Stable angina: The nature of exertional angina attacks does not change within 1 to 3 months, that is, the number of pain attacks per day and per week is approximately the same, the degree of exertion and emotional excitement that induces pain is the same, the nature and location of pain does not change with each attack, the pain time limit is 3-5 min, and the pain is relieved at the same time after rest or use of nitroglycerin. Worsening angina: Patients with stable angina who have experienced frequent changes in the frequency, degree, duration, and triggering factors of pain within 3 months are called worsening angina. This type can progress to myocardial infarction or sudden death, or it can return to stable angina pectoris. Primary angina and worsening angina are unstable and prone to acute events such as myocardial infarction, arrhythmia or sudden death, and are unstable angina.