Inflammation of the cornea is the clinical term for “keratitis”. The cornea is the convex, highly transparent membrane at the front of the eye that provides most of the refractive power for the eye. The cornea is transparent and non-vascular, but has abundant and sensitive nerve endings, so if something touches the cornea, the eyelids will involuntarily close to protect the eye. This is why keratitis is characterized by blurred vision and eye pain. The cornea is located at the front of the eye and is in direct contact with the outside world, making it susceptible to inflammation from microorganisms, trauma, and physical and chemical irritants. Currently, keratitis is clinically classified as infectious, immune, dystrophic, neuropathetic, and exposure based on the cause. Infectious keratitis mostly occurs in the central zone of the cornea, while immune keratopathy tends to occur in the peripheral part of the cornea. Among them, the most common is infectious keratitis. Infectious pathogenic microorganisms include bacteria (such as S. pneumoniae, S. aureus, Streptococcus hemolyticus, Pseudomonas aeruginosa, gonococcus, etc.), viruses (herpes simplex virus, adenovirus, etc.), fungi, Echinococcus amebae, mycoplasma, chlamydia, syphilis spirochetes, etc.