In our country, the majority of patients with liver cirrhosis are hepatitis cirrhosis, and some are alcoholic cirrhosis, autoimmune cirrhosis, etc. The energy metabolic status of the organism of these patients varies depending on the etiology. The metabolic catabolic state of patients with alcoholic cirrhosis is worse than that of patients with hepatitis B cirrhosis, and this difference is not just in fat and lean, but to assess the metabolic state of the three major nutrients in the body. 1, cirrhotic patients advocate a balanced diet, eating less and more meals. Advocate bedtime meal addition, not extra added, but a part of the dietary needs of the day set aside. The 50g staple-based intake in the bedtime meal addition meal is enough. This 50g of staple food can provide 200 calories, which can ensure the metabolic caloric demand during the night sleep, and this metabolic caloric demand can avoid the phenomenon of catabolism of protein and fatty substances in cirrhotic patients that we talked about earlier. If this is done over a long period of time, it will be seen that the protein consumed or supplemented over a long period of time will not be lost or reduced too much in a short period of time, meaning that the albumin level can be maintained. This also explains a clinical phenomenon: patients with hypoproteinemia, we always infuse protein, but always see protein elevation, the reason is that the patient we infuse protein as an energy supply material consumed, and if we implement additional meals, this phenomenon can be improved to a certain extent. 2, protein intake should be individualized, not the more you eat, the better. The protein consumption of all liver disease patients is extraordinary, and the metabolic breakdown of protein is limited. So if if if the liver is very good, intake more is likely to be hyperlipidemia; if the liver is not good, intake more is also still low protein, that is to say, poor utilization of the body, so, for patients with poor liver function, protein in the intestine outside the breakdown products can not be eliminated in a timely manner, clear, and may induce hepatic encephalopathy. Therefore, our intake is enough to meet the basic requirements. Meat, eggs, milk, fish, soy products are all proteins, fish or high-quality protein. 3, pay attention to vitamin and trace element supplementation, especially the B vitamins have a great impact on appetite. Some trace elements, such as zinc and selenium supplementation can accelerate the repair of damaged tissues, if lacking, then the repair of injured liver cells will be delayed. There is also a correlation between zinc and primary liver cancer, and patients with liver disease are zinc deficient, so we can supplement zinc, either medically or as a dietary nutrient. To have a long-term treatment regimen, we emphasize that dietary nutrition is consistent throughout. In this program, we need hepatitis patients to have no or few complications and to have a good status of life, quality of life. A balanced diet, for all patients with liver disease, is a long-term integrated treatment rather than a point. With long-term attention to dietary nutrition based on other comprehensive treatments, patients will have a higher quality of life and longer survival.