How to understand tricuspid valve insufficiency? Experts say that the tricuspid valve cannot close completely during systole due to congenital or acquired factors causing tricuspid valve lesions or dilatation of the tricuspid annulus, which is called tricuspid insufficiency. Tricuspid valve insufficiency can be functional or organic. The former is secondary to lesions that cause right ventricular dilation and has a high incidence, such as primary pulmonary hypertension, mitral valve lesions, pulmonary valve or funnel stenosis, and right ventricular myocardial infarction. The latter can be congenital anomalies such as Ebstein’s malformation and common atrioventricular channel, or acquired lesions such as rheumatic inflammation, coronary artery lesions causing tricuspid papillary muscle insufficiency, trauma, and infective endocarditis. Finally, experts point out that the prognosis of tricuspid valve insufficiency depends on the nature of the primary cause and the severity of heart failure, and that the prognosis is often worse for those with primary pulmonary hypertension and chronic pulmonary heart disease than for those with mitral valve lesions or atrial septal defects. Medical treatment of tricuspid valve insufficiency may relieve symptoms, but surgical treatment of tricuspid valve insufficiency can be curative.