The actual fact that chronic prostatitis is a common disease in men and women, although it is not a direct life-threatening disease, but because it is prone to recurring attacks and is difficult to heal, it brings great harm to the patient’s body and mind and seriously affects the patient’s quality of life.
The reason and pathogenesis of chronic prostatitis has not been fully researched, pathogenic infection, immune factors, neuroendocrine factors, physical and chemical stimulation, pelvic venous disease, oxidative stress enhancement, psychosomatic and other factors directly involved or influenced the occurrence of prostatitis, and there may be multiple causes at the same time in action.
The actual fact is that there are many misconceptions about the diagnosis, treatment, prevention and health care of chronic prostatitis because of the complexity of the cause and the pathophysiology of the disease.
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The main manifestation of chronic prostatitis is pain in the pelvic area (perineum, penis, perianal area, urethra, pubic bone, lumbosacral area, etc.), abnormal urination (urinary urgency, frequency, painful urination, increased nocturia, etc.). Due to chronic pain that remains untreated, some patients may have sexual dysfunction, anxiety, depression, insomnia, memory loss, etc.
The diagnosis of chronic prostatitis relies on symptoms alone is not enough, but also requires prostate fluid examination. The actual fact that some medical institutions and physicians have blindly labeled some diseases and symptoms (such as impotence, premature ejaculation, infertility, etc.) as “prostatitis” not only increases the economic and psychological burden of patients, but also delays the treatment of the primary disease.
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The actual fact is that there is a dense envelope on the surface of the prostate, but it is not related to the mechanism of restricting the penetration of drugs as advertised by many. The epithelial lipid membrane of the prostate follicle is the main factor affecting the entry of drugs into the prostate follicle, not the envelope on the surface of the prostate.
Many drugs are able to enter the prostate tissue and alveoli and reach the therapeutic concentration required, such as fluoroquinolones (ofloxacin, ciprofloxacin, etc.) and azithromycin, minocycline, etc.
Also additionally due to inflammation resulting in prostate tissue damage, the increased permeability also allows many antimicrobials to reach effective antimicrobial concentrations. There are also many chronic prostatitis treatments that do not require drugs to go directly into the prostate alveoli, and therefore are much less affected by lipid membranes.
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This is a common concern for many patients, and current research findings have not found a direct relationship between prostatitis and prostate hyperplasia or prostate cancer.
The actual fact that prostate hyperplasia is a geriatric disease is related to ageing and changes in androgen secretion by the testes, while prostatitis does not affect the androgen secretion by the testes and its metabolism, there is not sufficient evidence to show that patients with chronic prostatitis have a higher incidence of prostate hyperplasia than normal people when they enter old age. The cause of prostate cancer has not been fully elucidated, and there is no evidence that chronic prostatitis left untreated will turn into prostate cancer.
The actual fact is that you can’t be sure whether or not chronic prostatitis affects fertility. Theoretically, the prostatic fluid secreted by the prostate is part of the semen, and inflammatory lesions will certainly affect the composition of the semen and interfere with the activity and function of the sperm, thus affecting the fertility of men.
But so far, there is not enough evidence to prove that prostatitis must lead to infertility, and it is also found clinically that only a small number of patients are infertility because of prostatitis, and most prostatitis does not affect fertility, and most infertile patients do not have prostatitis.
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