The prevalence of hyperthyroidism in pregnancy is about 0.2%-2%. The prevalence is about 0.2% to 2%, and 95% of hyperthyroidism in pregnancy is due to Graves’ disease. The pattern of clinical manifestations is that the symptoms of hyperthyroidism increase in the early stages of pregnancy and decrease in the late stages. The effects of hyperthyroidism on pregnancy and fetus 1. The effects of hyperthyroidism on the mother during pregnancy: reproduction or aggravation of hyperthyroidism, menstrual disorders, amenorrhea, non-ovulatory menstrual cycle, and reduced chance of conception. The adverse effects of uncontrolled hyperthyroidism on pregnant women include miscarriage, preterm delivery, gestational hypertension syndrome, congestive heart failure, thyroid crisis, placental abruption and infection. 2. The effects of hyperthyroidism on the fetus during pregnancy include neonatal hyperthyroidism, intrauterine growth retardation, preterm infants, full-term small-like infants, risk of low birth weight, stillbirth, and fetal malformation. Effective control of hyperthyroidism can significantly improve the outcome of pregnancy. The relationship between hyperthyroidism in pregnancy and the incidence of congenital malformations is inconclusive. Studies have reported a high incidence of fetal malformations in patients with untreated hyperthyroidism and a low incidence in the group treated with ATD. However, it has also been reported in the literature that hyperthyroidism is not associated with fetal malformations. The clinical manifestations and diagnosis of hyperthyroidism in pregnancy are very similar to those of hyperthyroidism in terms of both hypermetabolic syndrome and physiological goiter. As the TBG increases, the blood TT3 and TT4 also increase accordingly. Hyperthyroidism should be suspected if the weight does not increase with the number of months of pregnancy, if the proximal extremities are thin, and if the HR is above 100 beats/minute at rest. If serum TSH decreases and FT3 or FT4 increases, hyperthyroidism can be diagnosed. If there is also infiltrative proptosis, diffuse goiter, tremor or vascular murmur in the thyroid area, and positive serum thyroid stimulating antibodies (TSAb), Graves’ disease may be diagnosed.