Pharmacological and/or surgical treatment is generally directed at the cause of the pathogenesis.
Because cerebral vasospasm can lead to a decrease in cerebral blood flow in its blood supply area, resulting in delayed ischemic neurological deterioration with symptoms such as hemiparesis, aphasia, and impaired consciousness, which can eventually lead to cerebral infarction, complications need to be actively prevented and treated.
Currently, commonly used anti-cerebral vasospastic drugs include nimodipine and fasudil, etc. When drug therapy is ineffective, endovascular interventional surgery is usually considered for ischemic symptoms.