Pharmacological and/or surgical treatment is usually administered to address the cause of morbidity.
As cerebral vasospasm can lead to a reduction in cerebral blood flow to its blood supply area, resulting in delayed ischaemic neurological deterioration with symptoms such as hemiparesis, aphasia and impaired consciousness, which can eventually lead to cerebral infarction, complications need to be actively prevented and treated.
Anti-cerebral vasospasm drugs such as nimodipine and fasudil are currently in common use. When drug treatment is ineffective, endovascular intervention is usually considered for ischaemic symptoms.