Hashimoto’s thyroiditis is a disease of the thyroid gland that is increasing in incidence year by year, has a long course and can have lifelong effects.1. The name Hashimoto’s thyroiditis is unique because Hashimoto’s thyroiditis was first reported by a Japanese physician and was later named after him, Hashimoto’s disease. The common population of Hashimoto’s thyroiditis, Hashimoto’s thyroiditis, has a high prevalence in women, the age of onset is young and middle-aged, and in recent years the onset is trending towards a lower age. The first thing you need to do is to get a good idea of what you are getting into. The first thing you need to do is to get a good idea of what you are getting into. The disease is now mostly considered to be a chronic inflammatory reaction and an autoimmune disease. 4. The clinical course of Hashimoto’s thyroiditis Hashimoto’s thyroiditis changes these manifestations because the antibodies destroy the cells of the thyroid gland. On the one hand, the large amount of T3 and T4 stored in the cells and not released into the bloodstream may produce the clinical manifestations of hyperthyroidism after cell destruction; on the other hand, as more and more cells are destroyed, the amount of T3 and T4 eventually tends to be depleted, i.e. hypothyroidism. Therefore, a typical Hashimoto’s thyroiditis goes through such a course: (1) Transient hyperthyroidism Hashimoto’s thyroiditis generally has a course of a few months, the vast majority of which have no clinical symptoms, not even an increase in FT4, this is related to the speed of destruction of the thyroid gland, the speed of destruction, the short period of release of more, it is easy to hyperthyroidism, high antibody test values may be suggestive. (2) Stable phase Hashimoto’s thyroiditis will occur after a transient hyperthyroidism, after all, the thyroid gland is also the kind of organ that is rich in reserves and it is not that easy to hypothyroidism. (3) Eventually, Hashimoto’s thyroiditis will go into hypothyroidism. The main point in the diagnosis of Hashimoto’s thyroiditis is the TPOAb in the thyroid antibody series, which is often tens of times higher, and the clinical diagnosis is often confirmed by a level higher than 400, and commonly higher than 1000. ECT imaging of the thyroid can be performed in targeted patients. If we understand that the manifestations of hyperthyroidism are at most transient, then anti-thyroid drugs are superfluous, surgery, iodine 131 are not considered, and ATD itself has no effect on the T3 and T4 already released into the blood. What does work? The beta-blocker, “Tylenol”, is sufficient. Patients with good A function can not intervene, and the method for autoimmune process has not been studied. If you are in hypothyroidism, you can replace the treatment with eugenol.