The loss of compensatory phase of cirrhosis after hepatitis B has two meanings: 1. The disease has now progressed to the stage of cirrhosis, and the cause of cirrhosis is hepatitis B virus replication. 2. Cirrhosis has reached an advanced stage and the function of the liver has lost its compensation. Because the liver is the main organ for synthesizing protein, the synthesis capacity of the liver decreases after cirrhosis, and there is a decrease in the level of albumin. The liver is an important place for the transformation and metabolism of various substances in the body, and cirrhosis will show abnormalities in the metabolism of bilirubin. Therefore, cirrhosis in the decompensated stage is characterized by a decrease in albumin and an increase in bilirubin in liver function. Cirrhosis is also associated with portal hypertension and esophagogastric fundic varices, which are prone to rupture and bleeding. After cirrhosis, the metabolism of blood ammonia and other toxic substances decreases, and hepatic encephalopathy is likely to occur. Therefore, whenever one of ascites, hepatic encephalopathy, or ruptured esophageal varices bleeds, it marks the liver has entered the decompensated stage.