Cirrhosis, as the name suggests, is a pathological change characterized by diffuse liver fibrosis, connective tissue hyperplasia, and pseudolobular tissue formation. The clinical diagnosis of cirrhosis often refers to the presence of hepatic impairment and portal hypertension, which is mainly manifested by esophagogastric fundic varices, splenomegaly, and ascites Cirrhosis is divided into compensated and decompensated stages. The liver has a strong regenerative capacity, when hepatocyte necrosis and. When the regenerated hepatocytes are in balance, it is able to maintain normal liver function, which is often referred to as the compensated phase of cirrhosis. This is the period when cirrhosis has occurred, or is progressing, but liver function tests are normal. After cirrhosis continues to progress into the decompensated stage, the overall number of hepatocytes decreases and the compensatory capacity exceeds the limit, or the number of regenerated stem cells is much less than the number of lost hepatocytes, at which time liver function [synthesis disorder] synthesis disorder occurs and various complications of cirrhosis can occur. For compensated cirrhosis, if and when the cause is removed by effective treatment, it is possible to reverse the trend of cirrhosis or even completely cure it, and some abnormalities of liver function can be completely restored. In summary, cirrhosis is divided into compensated stage and decompensated stage, and it is a gradual process, normal liver function may also have cirrhosis, may be in the compensated stage of cirrhosis, but the decompensated stage is certain to have abnormal liver function.