High blood uric acid does not necessarily lead to the development of gout; about 5-15% of patients with hyperuricemia will develop gout. Uric acid crystals are formed when blood uric acid is higher than normal. Uric acid crystals, if deposited in the kidneys, joints, and subcutaneous areas, will cause tissue damage and may lead to symptoms or signs such as acute gouty arthritis, gouty nephropathy, and gouty stones. Only 5-15% of patients with hyperuricemia will develop gout, and most patients can live their entire lives without experiencing gout symptoms. However, gout is basically caused by hyperuricemia, so hyperuricemia remains a high risk factor for gout. It is recommended that patients with hyperuricemia consult a doctor in a timely manner to control the consumption of alcohol and high purine food intake, if necessary, under the guidance of a physician should be reasonable use of allopurinol and other uric acid-lowering medication, do not self-medication.