Some hyperthyroid patients stop taking medication after their symptoms have completely disappeared or their “A” function has returned to normal, which is very inappropriate and prone to relapse. Generally speaking, the course of medication for hyperthyroidism needs at least 1.5~2 years, and if there is a family history or relapse of treatment, the medication time should be further extended. Do not stop the medication too early, and do not stop using it, otherwise it will easily lead to the relapse of hyperthyroidism. It is generally believed that the medication can only be stopped if the following conditions are met: (1) complete disappearance of hyperthyroid symptoms, reduction of thyroid gland, disappearance of vascular murmur and improvement of proptosis; (2) normalization of thyroid function (FT3, FT4, TSH) and negative thyroid stimulating antibody (TSAb); (3) small maintenance dose of medication (PTU25mg/day or MM2.5mg/day); (4) total course of treatment reaches more than two years. 2, not enough attention to adverse drug reactions, ignore the regular review of anti-thyroid drugs common adverse reactions are leukopenia, liver function damage and drug rash, and even life-threatening in serious cases. Since most of these adverse reactions occur within the first two months after the drug is administered, patients must be instructed to go to the hospital once a week for routine blood tests and liver function at the beginning of treatment, and once symptoms such as sore throat, fever and general malaise appear during the drug administration, they should immediately go to the hospital. Generally speaking, when the patient’s white blood cells are lower than 4×109/L and neutrophils are lower than 2×109/L, additional leukocyte-raising drugs (e.g. Lixisen, vitamin B4, etc.) must be administered. Note: Although shark’s liver alcohol can also raise white blood cells, it should not be used in patients with hyperthyroidism because of its high iodine content, which can lead to recurrence or aggravation of the disease) or with a small dose of prednisone, the white blood cells will generally recover quickly. If, after the above treatment, the white blood cell count is still below 3×109/L and the neutrophil count is below 1.5×109/L, accompanied by fever, sore throat, arthralgia and other symptoms of granulocyte deficiency, the patient must immediately stop taking antithyroid drugs and give granulocyte colony-stimulating factor, plus symptomatic treatment with effective broad-spectrum antimicrobial agents, and the patient should be sterilized and isolated if possible, otherwise it may lead to serious infection or even The patient should be isolated and disinfected if possible, otherwise it may lead to serious infection or even life threatening. For drug rash, anti-allergic drugs (e.g. paracetamol) can be added or replaced by other thioureas, but discontinuation is usually not necessary. If the rash is severe and deteriorates into exfoliative dermatitis, the medication should be stopped immediately and glucocorticoids should be administered. If the patient has abnormal liver function before taking anti-thyroid drugs (ATD), it means that the abnormal liver function is caused by hyperthyroidism itself and has nothing to do with ATD, so it can be treated with anti-thyroid drugs while taking liver-protective drugs. 3. Blind iodine supplementation without differentiating the cause Graves’ disease (also known as “diffuse goiter with hyperthyroidism”) and iodine-deficient goiter (also known as “endemic goiter”) can both lead to goiter, but the causes of the two are completely different. The former is related to genetics and autoimmunity, while the latter is due to compensatory hyperplasia of the thyroid tissue caused by insufficient iodine intake. In order to reduce the synthesis of thyroid hormone, hyperthyroidism patients should have a low iodine diet, preferably with non-iodized salt for stir-fry, and iodine-rich seafood such as seaweed, kelp and seafood should be eaten sparingly or not at all, otherwise the recovery from hyperthyroidism will be slow and easy to relapse. In recent years, it has been found that amiodarone, an anti-arrhythmic drug, is an important factor in increasing the incidence of hyperthyroidism in the elderly, as each 100mg of amiodarone contains 37.2mg of iodine. 4. Simply pay attention to medication and neglect physical and mental recuperation Hyperthyroidism makes the body in a high metabolic state and consumes a lot. Therefore, hyperthyroidism patients must pay attention to rest, avoid overwork and increase nutrition. In addition, hyperthyroidism can be triggered by high mental tension, excessive stress, serious infections, and the consumption of strong tea, coffee, tobacco and alcohol. Therefore, hyperthyroidism patients must pay attention to maintain emotional stability, peace of mind and good sleep, especially in the early stage of the disease, it is best to rest in bed or hospitalization. 5. It is believed that if you have hyperthyroidism, you cannot get pregnant, and once you get pregnant, you have to stop taking your medication. Pregnancy does not usually lead to significant deterioration of hyperthyroidism, so hyperthyroidism is not an absolute contraindication to pregnancy. It is generally recommended that patients with hyperthyroidism should get pregnant after the disease is cured and the medication is completely stopped. However, if the patient’s condition is well controlled at this stage and only a small dose of medication is needed for maintenance, pregnancy is also allowed. It is generally believed that this will not increase complications for the mother and the baby during pregnancy and the prognosis for the mother and the newborn is good. On the contrary, if the hyperthyroidism is not effectively controlled, pregnancy is not advisable. Otherwise, it will easily cause miscarriage and premature delivery, and not only that, because the pregnant woman is in a high metabolic state, she cannot provide sufficient nutrition and oxygen to the fetus, which may also lead to fetal growth retardation and intrauterine distress. In terms of medication, pregnant women with hyperthyroidism should choose propylthiouracil instead of tabazol, as the former has a larger molecular weight when combined with proteins in the pregnant woman’s body, which does not easily pass through the placenta and enters the fetal blood in a smaller amount and does not affect the fetus. In addition, during pregnancy, thyroid function needs to be closely monitored and the dose of propylthiouracil should be adjusted in time so that the serum FT3 and FT4 levels are at 1/3 of the normal high limit. It is important not to overdose and cause hypothyroidism, which can affect fetal brain development. Since antithyroid drugs (ATD) can be secreted from breast milk and affect fetal thyroid function, patients with hyperthyroidism should not breastfeed when taking ATD therapy. Some women with hyperthyroidism simply do not take any medication during pregnancy because they are concerned about the side effects of the medication, resulting in uncontrolled hyperthyroidism, which has serious adverse effects on both themselves and the fetus. In addition, if a patient has previously received radioactive iodine treatment, pregnancy should be prohibited for 6 months after treatment to reduce the risk of fetal teratogenicity.