Uterine adhesions are a phenomenon recognized at the end of the 19th century and can be traced back to its first description by H. Fritsch in 1894.
Asherman studied uterine adhesions in depth and defined them as: intrauterine adhesions due to trauma or narrowing above the internal cervical opening. Uterine adhesions are usually due to damage to the uterine mucosa. The causes of uterine adhesions are varied and are usually post-pregnancy adhesions and non-pregnancy intrauterine adhesions. The main causes of post-pregnancy adhesions are: scraped or unscraped uterus after abortion, post-partum scraping, post-abortion or post-partum endometritis, local ischemia of the uterus due to post-partum bleeding, and uterine artery embolization. Intrauterine adhesions after non-pregnancy include adhesions after hysteroscopic surgery (e.g., fibroid removal, endometrial polyp removal, endometrial septum removal, hyperplastic endometrial removal, etc.) and uterine adhesions after genital tuberculosis infection. The incidence of uterine adhesions is on the rise. A total of 1250 cases of uterine adhesions were reported in the literature during a total of 88 years from 1894 to 1982, while a total of 2500 cases of uterine adhesions were reported in the literature during a total of 26 years from 1982 to 2008. The rapidly increasing incidence is undoubtedly associated with the use of diagnostic hysteroscopy and 3D ultrasound, which are the best tools for the diagnosis of uterine adhesions. Some uterine adhesions are very complex and severe and can lead to amenorrhea, recurrent miscarriage, infertility, placenta previa or implantation. The treatment of these complex uterine adhesions requires several hysteroscopic procedures. How many hysteroscopic procedures are appropriate and what is the impact on reproduction? This has been studied retrospectively by several experts in France. The literature was published in Fertility
and Sterility October 2012.