These include true ventricular wall tumors and pseudoventricular wall tumors. True ventricular wall tumors occur in more than 95% of patients with coronary artery disease due to transmural myocardial infarction, in which the entire myocardium in the infarcted area is necrotic and gradually replaced by fibrous scar tissue, resulting in dilatation and thinning of the ventricular wall, outward expansion of the thin layer of the ventricular wall, and loss of mobility or paradoxical motion of the heart during contraction. Pseudoventricular wall tumors most often arise 5 to 10 days after acute myocardial infarction, mostly due to occlusion of the gyral branch, and are caused by the rupture of the left ventricle wrapped by the adhesions of the outer pericardium, whose wall does not include myocardial tissue and is composed of a blood clot, epicardium, and partially adherent pericardium.