Concept: Ventricular septal defect is a common congenital heart malformation, mostly a single malformation, accounting for about 20% of congenital heart disease; it can also be a component of compound heart malformations, such as tetralogy of Fallot, complete atrioventricular channel, right ventricular double outlet, complete transposition of the great arteries, pulmonary atresia, etc. Ventricular septal defect is a condition in which the ventricular septum is hypoplastic during embryonic life and abnormal traffic forms between the left and right ventricles, producing a left-to-right shunt at the ventricular level. The defect is often 0.1-3 cm and most are located in the membranes, less frequently in the subpulmonary ventricular defect, the bicuspid subarterial ventricular defect, and the myocardial ventricular defect. If the defect is <0.5 cm, the shunt flow is small and it is a restrictive ventricular septal defect with no obvious clinical symptoms. If the defect is small, the heart size can be normal, but if the defect is large, the left ventricle is larger than the right ventricle, which is prone to cardiac insufficiency, affecting the growth and development of the child, and even leading to recurrent colds, fever and pneumonia. Etiology: 1. Genetic factors (20%): There is a family history of congenital ventricular septal defect. It is not uncommon for siblings to have the disease at the same time, or for parents and children to have the disease at the same time, and the nature of the disease is very similar. If the first child born to the mother has the disease, the likelihood of the second child having the disease is about 2%; if two consecutive children are born with ventricular septal defect, the number of children born with the disease may increase to 10%. If the mother has the disease, the risk of the second child having the disease is 10%. 2. Pregnancy disease factor (18%): Pregnant women with untreated and uncontrolled diabetes mellitus may have a significantly higher risk of congenital ventricular septal defect, but the risk decreases if the disease is controlled and stabilized in early pregnancy. Physical and chemical factors (15%): Exposure to teratogenic drugs such as lithium, phenytoin sodium or steroids in early pregnancy can lead to an increase in fetal morbidity. Excessive exposure to radioactive substances such as X-rays and isotopes in early pregnancy. Viral infections. If a woman is infected with a virus during the first three months of pregnancy, especially during the third to eighth weeks of pregnancy, the fetus is susceptible to the disease. 4, other factors (15%): such as smoking and alcohol consumption by both parents during conception can induce congenital ventricular septal defect. Diet care: 1, daily life should pay attention to reasonable nutrition, food as diverse as possible, eat more high protein, multivitamin, low animal fat, easy to digest food and fresh fruits and vegetables. 2, do not eat stale and spoiled or irritating things, less fumigation, baking, pickled and soaked, fried, over-salty food, staple food coarse and fine grains with to ensure nutritional balance.