The emboli of cerebral thrombosis have the following sources: 1, atherosclerotic plaque shedding, due to hypertension, diabetes mellitus, dyslipidemia and other cerebrovascular disease risk factors poorly controlled for a long time, causing cerebral atherosclerotic lesions, which can promote platelet adhesion and aggregation in the blood and then form thrombus. After the unstable thrombus is dislodged, it can reach and block the distal blood vessels with blood flow, leading to cerebral thrombosis. 2, heart disease is also a common source of cerebral thrombosis, combined with heart disease, especially in patients with previous atrial fibrillation, atrial flutter, heart valve disease or artificial heart valve disease, the inner wall and valves of the heart can form thrombus or redundancy, and these thrombus or redundancy can block the cerebral arteries after they are dislodged, which can also form cerebral thrombosis. Other causes, such as fracture can cause fat emboli, deep vein thrombosis in the lower extremities, and tumor patients in a hypercoagulable state to form emboli, can reach and block the distal vessels with blood flow, leading to cerebral thrombosis. In general, risk factors such as blood pressure, blood sugar and blood lipids should be actively controlled. Patients with atrial fibrillation or valvular disease should apply anticoagulant drugs correctly to prevent the formation of thrombus from other causes and minimize the risk of cerebral thrombosis.