What is the difference between spastic cerebral palsy and torsional spasticity?

  Torsionspasm (TS), also known as deformational dystonia, torsional dystonia, or dystonia of the nucleus pulposus, is characterized by involuntary and repeated abnormal synchronous contractions of the active and antagonistic muscles, resulting in torsional movements or abnormal posture of the affected limb. The symptoms of TS may be confined to one part of the body, or may involve multiple parts or half of the torso, or even the whole body.  The difference between spastic cerebral palsy and torsional spasm Spastic cerebral palsy is the most prevalent type of cerebral palsy in clinical practice, accounting for 60% to 70% of all patients, and the most typical symptom of patients is tiptoeing and scissor gait when walking.  From a medical point of view, patients with spastic cerebral palsy are characterized by hyperactive detrusor reflexes and persistent muscle tension causing motor dysfunction. The degree of muscle tension is influenced by various conditions, such as the intensity of the stimulus and the excitability of the affected child. If the muscle tension is increased, the muscles around the impaired area, such as the scapula and hip joint, contract strongly and simultaneously, and a typical fixed posture can appear. Weakening of a particular group of muscles may occur due to the opposite neuroinhibitory barrier that antagonizes the muscle spasm.  The spasticity is often aggravated by exertion and agitation and relieved when the patient is quiet and asleep. Voluntary movements are very difficult due to joint spasticity. The hemiplegic patient has a circular gait, which requires elevation of the pelvis when walking because of the difficulty of flexion due to the extension of the affected side. The lower limbs rotate outward in a semicircular motion.  The cause of torsional spasm is similar to that of spastic cerebral palsy. It is mainly due to jaundice caused by premature birth, asphyxia, hypoxia, fever, etc., which causes incomplete brain development and increases the muscle tone of the limbs, twisting the head to one side, twisting the upper limbs to the back side and twisting the lower limbs to the outside. It is also a symptom of repetitive involuntary movements and abnormal twisting posture caused by the uncoordinated, intermittent and continuous contraction of the synergistic and antagonistic muscles of the skeletal muscles of the body, so it is also called dystonia syndrome.  The child shows nervousness and fear, sweating, cervical, facial, and pharyngeal muscles may be involved in the attack of torsion spasm, which may lead to slanting neck, facial muscle spasm, swallowing and dysarthria.  The common manifestations of twisting spasm are: head twisted to one side when standing, shoulders tilted back, one arm stretched forward and one stretched backward, both knees bent inward, both feet separated to maintain balance; or accompanied by foot inversion, the soles of the feet can not be completely on the ground. The patient’s body will start to be bowed when lying down, supported by the shoulders and hips, and some can only lie prone on the bed. After a long time, certain parts of the muscles may be abnormally hypertrophied and the joints may be contracted and deformed, and the symptoms will disappear after falling asleep.  In addition to the above differences, spastic cerebral palsy is also very different from torsional spasticity in terms of treatment methods.  The most suitable treatment method for patients with spastic cerebral palsy is FSPR surgery. By comprehensively adjusting the patient’s muscle tone so that the muscle tone of the spastic muscles is as close to normal as possible, it can provide a long-term, stable and complete solution to the pain of the patient’s muscle spasms and provide the prerequisite for the maximum recovery of their motor functions.  In addition, the FSPR procedure has the unparalleled advantage of other surgeries in that it selectively blocks part of the posterior nerve root fibers without affecting the anterior nerve roots that govern muscle movement and motor function. The exact site of surgery can be determined by the patient’s specific condition: surgery in the lumbar spine can address lower extremity spasticity, and surgery in the cervical spine can address upper extremity spasticity.  In contrast, patients with torsional spasticity should undergo carotid epicranial sympathetic dissection, and intracranial vascular disease should be excluded by cranial MRA prior to surgery. After intraoperative general anesthesia with tracheal intubation, the patient is placed in a supine position with shoulder pads and head tilted back. A longitudinal incision of approximately 75 px. in length was made on the medial edge of the inferior and middle sternocleidomastoid muscles bilaterally. The skin, broad cervical muscle and anterior cervical fascia were incised and the sternocleidomastoid muscle was pulled outward. The sternocleidomastoid muscle and sternocleidomastoid muscle were subconsciously separated, and the carotid artery sheath was medially retracted to expose the carotid artery sheath, and part of the carotid artery sheath was opened and excised; the common carotid artery was separated from the internal jugular vein gap, and special care was taken not to injure the vagus nerve between the two in this procedure.  The common carotid artery is then circumcised under the microscope for 2 to 75 px, and the vagus nerve is freed and the connective tissue around the nerve is excised. Thorough hemostasis is achieved, the muscle is sutured in layers, and the skin incision is then glued with adhesive tape. Through this surgical method, followed by corresponding rehabilitation training, it has played an outstanding role in improving the patient’s bilateral upper limb movement, speech, swallowing, chewing and other functions; it also improves the brain blood supply and oxygenation, etc.