Eyelid laxity is a specific type of eyelid disease that occurs in adolescents and is characterized by recurrent episodes of edema, thinning, wrinkling, and skin laxity of the eyelid skin, of unknown etiology, and can occur in one or both eyes. Eyelid laxity differs from eyelid skin laxity in the elderly in that, in addition to eyelid skin laxity, it is often accompanied by lacrimal gland prolapse, ptosis, and shortening of the transverse diameter of the eyelid fissure. Lacrimal gland prolapse is due to the weakness of the orbital septum; ptosis is mostly mild, and the strength of the levator muscle is usually good, but in some patients, the levator tendon membrane can be separated from the eyelid plate; the shortening of the transverse diameter of the eyelid fissure is due to the laxity of the canthal ligaments, and can be manifested as a blunt and rounded deformity of the outer corner of the eyelid canthus. It should be noted that in some patients, the shortening of the transverse diameter of the lacrimal fissure may conceal the ptosis, which will be manifested after shortening and fixation of the lateral canthal ligament during the operation. Therefore, the design of the surgical plan should be comprehensive and reasonable. Eyelid laxity is usually categorized into two types: hypertrophic and atrophic, with hypertrophic surgery focusing on removing prolapsed fat and reinforcing the orbital septum; and atrophic surgery focusing on removing excess skin. At the same time, the patient’s complications, such as lacrimal gland prolapse, ptosis and shortening of the transverse diameter of the eyelid fissure, are also addressed.