Clinically, scars can be classified into the following types according to their histology and morphology. Nuclear medicine dressing treatment is effective for superficial scars, hyperplastic scars and keloids! I. Superficial scars Superficial scars are formed by mild abrasions on the skin, or due to superficial second-degree burns, or superficial infections on the skin, usually involving the superficial layers of the epidermis or dermis. Clinical manifestations: rough surface, sometimes with pigmentation changes. The area is flat, soft and sometimes poorly defined from the surrounding normal skin. There is usually no functional impairment and no special treatment is needed. Proliferative scars may be formed whenever the injury involves the deep dermis, such as deep second-degree burns, cutting wounds, infections, the donor area after cutting medium-thick skin pieces, etc. Clinical manifestations: The scars are significantly higher than the surrounding normal skin, with local thickening and hardening. In the early stage, the scar surface is red, flushed or purple-red due to capillary congestion. At this stage, itching and pain are the main symptoms, and even scratching may cause the surface to break down. After a considerable period of time, the congestion decreases, the surface color becomes lighter, the scar gradually becomes softer and flatter, and the itchiness and pain are reduced or disappear. Generally speaking, children and young adults have a longer proliferation period, while older people over 50 years old have a shorter proliferation period; the proliferation period of keloid scars that occur in areas with rich blood supply, such as the face, is longer, while the proliferation period of keloid scars that occur in areas with poor blood supply, such as the ends of the limbs and the anterior tibial area, is shorter. Although the hyperplastic scar can be more than 2 cm thick, it does not adhere tightly to the deep tissue and can be pushed, and there is usually a clear boundary with the surrounding normal skin. The contractility of hyperplastic scars is less than that of contracted scars. Therefore, hyperplastic scars that occur in non-functional areas generally do not cause serious dysfunction, while large hyperplastic scars in joint areas can cause dysfunction due to their thick and hard splinting effect, which hinders joint movement. The hyperplastic scar located on the flexor surface of the joint may contract more obviously in the late stage, resulting in obvious dysfunction such as maxillofacial neck adhesions. Atrophic scars Atrophic scars, which involve the whole skin and subcutaneous fatty tissue, can occur after large third-degree burns, long-term chronic ulcers healing, as well as after electric shock injuries to areas with little subcutaneous tissue, such as the scalp and anterior tibial area. Clinical manifestations: The scar is hard, flat or slightly above the skin surface, and closely adheres to deep tissues such as muscles, tendons, and nerves. The scar has very poor local blood circulation and is light red or white in color. The epidermis is extremely thin and cannot withstand external friction and weight-bearing, so it can easily break down and form a chronic ulcer that does not heal. If it is healed for a long time, there is a possibility of malignant transformation in the late stage, and it is mostly squamous epithelial carcinoma in the pathology. Atrophic scars have great contractility and can pull the neighboring tissues and organs, causing serious functional disorders. The occurrence of keloid scars has obvious individual differences. Most keloid scars occur unobstructed within 1 year of local injury, including surgical procedures, lacerations, tattoos, burns, injections, animal bites, inoculations, acne and foreign body reactions, and many patients’ primary medical history may be forgotten. Clinical manifestations: The clinical manifestations of keloid scars vary widely. They generally appear as persistent growing lumps above the surrounding normal skin and beyond the original injury site, hard to the touch, poorly elastic, locally itchy or painful, with a pink or purplish surface in the early stages and pale white in the later stages, sometimes with hyperpigmentation and a more distinct boundary with the surrounding normal skin. The lesions vary in size from 2-3 mm papule-like to large palm-like patches. The morphology is diverse, ranging from relatively flat, symmetrical protrusions with right regular margins to unevenly elevated masses with irregular protrusions, sometimes growing like crab feet infiltrating into the surrounding tissue. Its surface is an atrophic epidermis, but the epidermis of keloids within the earlobe can be close to normal skin. Most cases are solitary, with a few cases being multiple. The keloid develops rapidly within a few weeks or months after the injury and can grow continuously and continuously or remain stable for a considerable period of time. Inflammatory necrosis may develop within the lesion due to residual follicular glands or liquefied necrosis due to central ischemia. Keloid scars generally do not undergo contracture and do not cause functional impairment, except for a few lesions in joint areas that cause mild restriction of movement. Keloid scars generally do not degenerate on their own; occasionally, lesions have been reported to degenerate after menopause, independent of their course, location, etiology, or symptoms. Malignancy of keloids has been reported, but the incidence is very low. V. Others Clinically, according to the morphology of keloid scars, they can be divided into several types such as linear keloid scars, webbed keloid scars, depressed keloid scars, and bridge-shaped keloid scars.