Problem skin usually refers to skin with disfiguring diseases, such as acne, seborrheic dermatitis, cosmetic-induced contact dermatitis, hormone-induced hormone-dependent dermatitis, etc. It is important to go to a regular hospital to see a dermatologist to get effective treatment and guidance. Take hormone-dependent dermatitis, the same area, long-term topical corticosteroids, hormones by interfering with the differentiation of the epidermis, inducing changes in skin structure and function, keratin-forming cells proliferation is inhibited. This leads to a decrease in the formation of hyaline stratum corneum particles and eventually to a thinning of the stratum corneum. Impaired epidermal barrier function leads to increased transepidermal water loss and increased sensitivity of the skin to external stimuli. Resulting in thinning of the epidermis and dermis. It causes hypopigmentation and hyperpigmentation, or alternatively, hypopigmentation and hyperpigmentation may alternate, showing florid changes. Due to the decrease in the number of layers of the stratum corneum, less melanin migrates to the keratin-forming cells, causing hypopigmentation. Hyperpigmentation may be related to the activation of melanocytes by glucocorticoids to regenerate pigment. Presence of vascular manifestations. Weakened adhesion between collagen fibers in the vessel wall may lead to widening of the vessels and loss of dermal collagen leading to surface vascularization. There is also rosacea/acne-like dermatitis. In hormone-induced rosacea-like lesions, there is a significant increase in the density of follicular worm mites, which close the follicular sebaceous outlet and act as carriers, causing an inflammatory response or metaplasia, and potent hormones can also proliferate the sebaceous glands, leading to the characteristic rosacea-like rash. Hormones can degenerate the epithelium of the hair follicle, causing the outlet to be blocked and an acne-like rash to appear or aggravate existing acne. Folliculitis can also occur. Infection of hair follicles and aggravation of pre-existing folliculitis can occur due to the immunosuppressive effects of hormones. Finally hormone dependence. Hormones have powerful anti-inflammatory properties and can suppress many dermatological symptoms, such as inhibiting the development of papules and reducing itching, vasoconstriction, and disappearance of erythema. However, hormones cannot eliminate the cause of the disease and can often cause aggravation of the original disease after discontinuation, which can lead to rebound phenomena such as inflammatory edema, redness, burning sensation, discomfort, and acute pustular rash. This phenomenon often occurs 2 to 10 days after discontinuation of hormones and lasts for a few days or about 3 weeks. The rebound phenomenon causes patients to continue to use hormones topically, resulting in hormone dependence. At present, there is no ideal treatment for hormone dependent dermatitis, but the main treatment is to gradually reduce the strength and dosage of hormone drugs, together with antibacterial and anti-inflammatory drugs, until all hormone drugs are withdrawn, the treatment process takes 1-2 years, which is difficult for most patients to accept. In the treatment, doctors and patients cooperate in order to recover and heal faster and better! Patients should be fully aware of the effects and side effects of hormones, be fully prepared mentally, be sure to be calm, maintain a good state of mind, and have firm confidence in overcoming the disease! Avoid eating, such as spicy, stimulating, fish, shrimp and seafood hair products; keep a happy mood and treat skin lesions positively; prevent wind and sunshine and other climatic environment stimulation and injury to the skin.