Cirrhosis is a common chronic diffuse progressive disease, which is caused by a variety of causes of chronic extensive liver parenchymal damage. Extensive degeneration, necrosis, and atrophy of hepatocytes are replaced by proliferating fibrous tissue, and normal liver lobular structures are destroyed, causing the liver to gradually harden and become cirrhotic. Cirrhosis is a chronic sclerotic disease of the liver caused by one or more pathogenic factors such as malnutrition, chronic alcoholism, viral hepatitis, intestinal infections, toxic effects and other long-term or repeated damage to the liver tissue. The onset and course of cirrhosis are slow. In the early stage of cirrhosis, patients may have loss of appetite, fatigue, nausea, aversion to oil, discomfort in the liver area, or no obvious clinical symptoms, and most of the liver function is normal or mildly abnormal; later, the liver gradually hardens and its function decreases significantly, manifesting as gastrointestinal symptoms, malnutrition, bleeding tendency, edema, endocrine disorders, jaundice, splenomegaly, ascites formation, and even hepatic coma. In addition, complications such as upper gastrointestinal bleeding, hepatic encephalopathy, primary hepatocellular carcinoma, infections and hepatorenal syndrome may occur.