Myocardial bridges are caused by abnormal development of the coronary arteries, and most patients with myocardial bridges do not experience any symptoms. A few patients may experience angina-like symptoms such as chest tightness, palpitations, arrhythmia, and myocardial infarction. Beta-blockers such as Betalucil (metoprolol tartrate) and calcium channel blockers such as verapamil can be used under medical supervision to relieve symptoms. Beta-blockers and non-dihydropyridine calcium antagonists can reduce the pressure on the wall coronary arteries and increase the filling time of the coronary arteries by decreasing myocardial contractility, slowing down the heart rate, and lengthening the diastolic phase, thus improving coronary arteries and myocardial perfusion, and reducing the occurrence of myocardial ischemia. When patients are diagnosed with myocardial bridges and clinical symptoms appear, they should be treated in a standardized manner under the guidance of a doctor, and should not use medication on their own, so as to avoid delaying the condition and causing adverse consequences.