In mild IgA nephropathy, if the condition can be controlled by active and standardized treatment, hematuria can be improved and may not continue to develop; if the condition continues to worsen without active and standardized treatment, hematuria will continue to develop. Due to the different conditions of individuals, whether hematuria develops or not, there are some differences. IgA nephropathy is a chronic glomerulonephritis caused by the abnormal deposition of immunoglobulin A in the glomerular tunica albuginea region, which pathologically manifests as tunica albuginea hyperplasia and the deposition of immune complexes in the tunica albuginea region with IgA as the main component. Most of the mild IgA nephropathy after active standardized treatment, hematuria can be slowly improved, usually will not continue to develop. At the same time, there is still a part of the population without active standardized treatment will recur hematuria, prone to mild membranoproliferative glomerulonephritis, which can lead to hematuria continue to develop. It is recommended that patients with IgA nephropathy go to the hospital in time and receive standardized treatment under the guidance of the doctor.