Tibial tuberosity epiphysitis, also known as tibial tuberosity osteochondrosis, tibial tuberosity osteochondritis, and aseptic necrosis of the tibial tuberosity epiphysis. From a variety of etiologic nomenclature, the pathogenesis, pathological essence of the understanding is not uniform, foreign literature is mostly referred to as Osgood-Schlatter disease, was reported by Osgood and Schlatter respectively in 1903 at the same time. Etiology It is generally believed that the tibial tuberosity epiphysis occurs as a result of acute or repeated chronic injury under the pull of the patellar tendon. The epiphysis is the center of skeletal development during adulthood, and the tibial tuberosity epiphysis is located on the proximal side of the tibia, anterior to the point of attachment of the patellar tendon of the quadriceps muscle. Epiphyseal inflammation of many epiphyses throughout the body occurs almost exclusively during the adult phase of development, and abnormal epiphyseal development should be the basis for the development of epiphyseal inflammation and epiphyseal osteochondrosis. Clinical manifestations Prevalent in adolescent boys, 11-15 years old, mostly with accelerated development and a preference for sports, and may have a history of strenuous exercise or trauma. Pain at the tibial tuberosity, aggravated by activity. There may be localized swelling, pressure pain, or even redness and heat in the tibial tuberosity. Active knee extension, aggravated by passive knee flexion or squatting, is caused by the patellar tendon pulling on the epiphysis. MRI may show patellar tendonitis or subpatellar bursa. The diagnosis is not difficult, but attention should be paid to differentiate it from osteosarcoma, which is a primary malignant bone tumor, and the proximal tibia is also a favored site, and adolescence is also a favored period. Treatment The disease is self-limiting, i.e. it heals on its own without medication, and patients are only instructed to take rest and limit knee activities, avoid running, jumping, bouncing, and long walks. In case of acute attacks, external fixation with a plaster can be used. Local closed treatment with glucocorticoids, although it can quickly relieve pain, can lead to tissue degeneration and necrosis, spontaneous rupture of the patellar tendon, and is not recommended.