The normal testicular sphincter contains a small amount of fluid that acts as a glide, enabling the testicle to slide in the scrotum. Under normal circumstances, the secretion and absorption functions of the sphincter wall are relatively stable. If lesions occur in the sphincter or in the organs surrounding the tissue, so that the secretion and absorption functions of the sphincter are out of balance, and when secretion is greater than absorption, it can lead to syringomyelia. If the sphincter effusion is more prolonged, the long-term increase in intrasphincter pressure will compress the testes and compress the spermatic cord to affect testicular ischemia, leading to poor testicular spermatogenesis and affecting fertility. Testicular syringomyelia secondary to tuberculosis, orchitis and other diseases, tuberculosis, orchitis and other diseases themselves affect the spermatogenic function of the testes, which is even more detrimental to fertility. Huge syringomyelia affecting conjugal life may also lead to infertility.