Ascites is one of the most common complications of decompensated cirrhosis, and in severe cases, pleural fluid, inguinal hernia, umbilical hernia, which seriously affects the quality of life of patients. Causes: portal hypertension, hypoproteinemia, impaired lymphatic return, increased antidiuretic hormone (aldosterone), hepatorenal syndrome, etc. Infection is often the causative factor. Clinical manifestations: abdominal distension, breath-holding in the presence of pleural fluid (mostly on the right side). There may be no conscious symptoms in a small amount of thoraco-abdominal fluid. With the increase of abdominal water, abdominal distension will gradually increase, especially after eating. Treatment: 1, antagonism of aldosterone, commonly used spironolactone, while potassium-protective diuretics, disadvantages of long-term use will appear male breast development, can be relieved after discontinuation of the drug, and also can be used alternately with aminoglutethimide. 2, diuretic use: furosemide, torasemide, hydrochlorothiazide. Diuretic effect is good, but all excrete potassium, generally used in combination with spironolactone, need to supplement potassium when urine volume increases significantly, and pay attention to monitor blood pressure, need to be used under the guidance of a doctor. 3.Correct hypoproteinemia, supplemental albumin, here is the intravenous input. 4.Reducing portal vein pressure commonly used are propranolol (price is too low, not good to buy); short-acting preparation of octreotide is limited to use during hospitalization, long-acting preparation is too high, and it is difficult for general patients to pay up. 5, the presence of infection anti-infective treatment, chest fluid infection while adequate drainage. 6, ascites reflux, good effect for treatment of intractable massive ascites. 7, protection of renal function. 8.Interventional treatment TIPS (portal venous shunt). 9.Chinese medicine.