The effect of thoracic spine lesions on the heart nerves: Various causes of different degrees of changes in the thoracic spine can compress and stimulate the innervation system of the heart, mainly the sympathetic nervous system, causing sympathetic nerve dysfunction, thus causing coronary artery spasm, myocardial ischemia, cardiac arrhythmia, abnormal heart activity, causing a series of clinical symptoms of heart disease. Its degeneration causes sympathetic nerve dysfunction, and the main lesions are as follows: 1. Thoracic disc herniation The intervertebral disc undergoes great stress in the movement and load of the spine, and with age, the disc gradually degenerates, the water content of the fibrous ring and the nucleus pulposus gradually decreases, the nucleus pulposus loses elasticity, the fibrous ring gradually fissures, and on the basis of degeneration, strain accumulation and external forces, the disc ruptures, and the nucleus pulposus The nucleus pulposus, fibrous ring and even the end plate protrude forward and backward, thus stimulating and compressing the nerve to produce symptoms. 2, ligamentum flavum hypertrophy ligamentum flavum is the spinal canal to maintain the stability of the spine of a primary tissue structure, in the spine degeneration, ligamentum flavum is subject to increased stress, its elastic fibers will degeneration or rupture, and long-term injury and repair process is bound to make ligamentum flavum hypertrophy, fibrosis, and finally lead to calcification of the primary belt. The result of the hypertrophy and calcification of the yellow primordial ligament is the narrowing of the spinal canal, which leads to nerve irritation and compression symptoms. 3, spinal stenosis spinal stenosis is caused by various causes of the shortening of the diameter of the spinal canal, compression of the dural sac, spinal cord, nerve roots and thus produce symptoms. The main causes include congenital developmental malformations, osteophytes, hypertrophy of the ligamentum flavum, ossification of the posterior longitudinal ligament, intervertebral disc lesions, post-traumatic vertebral fractures, slippage, and vertebral instability. 4, spinal instability spinal instability refers to the process of vertebral degeneration, the vertebral structure can not maintain its physiological balance and lead to vertebral body displacement beyond its physiological limits and the corresponding clinical symptoms, the factors that cause spinal instability are congenital developmental abnormalities, trauma, vertebral body degeneration, small joint pro disorder, local inflammation, etc. 5, thoracic vertebral body inflammation when the front of the vertebral body inflammation or tuberculosis, inflammatory mediators stimulate the thoracic sympathetic nerve, resulting in sympathetic edema, increased excitability, resulting in a series of clinical symptoms. 6, thoracic compression fractures into wedge-shaped changes, vertebral compression fractures are mostly caused by trauma, but also seen in the elderly osteoporosis, vertebral metastases, trauma and other cases. Compression fractures of the vertebrae can cause spinal instability and also cause spinal cord injury, edema, abnormal discharge, and excitation of the thoracic sympathetic nerve through the gray and white traffic branch, causing cardiac symptoms. 7, scoliosis, rotation and distortion of the spine The thoracic spinal cord is in a moderate volume balance in the thoracic spinal canal, the fat gap in the spinal canal is relatively small, coupled with the relatively tight fixation of the membranous ligaments, scoliosis causes the spinal cord to be in tension on one side and the spinal cord on the other, causing spinal nerve discharge and excitation of the thoracic sympathetic nerve, resulting in a series of clinical symptoms. When the thoracic spine is rotated and twisted, the thoracic spinal cord is also twisted due to the fixation of the membranous ligament, and the discharge occurs, which excites the thoracic sympathetic nerve through the conduction of the gray and white traffic branch, thus affecting the function of the innervated internal organs.