Everyone in life is more or less likely to have encountered the experience of eyelid jumping, the so-called left jumping money right jumping disaster, but there are some people jumping is a few years or even a dozen years, and with more and more serious, this situation should be considered facial muscle spasm. The rapid twitching (several times per second) of the muscles around the eyes and cheeks during the attack causes muscle spasms that narrow the eye fissures and tilt the corners of the mouth to the diseased side for a few seconds to tens of seconds, then relieves itself, and in severe cases the broad neck muscles also twitch at the same time. Facial muscle spasms can recur within a short period of time or stop for a longer period of time. Excitement, emotional stress, and conscious movement of the face may induce spasm attacks. The disease progresses slowly and usually does not get better spontaneously. Many patients with facial muscle spasms choose Botox to relieve them in the early stages of the disease, and when it comes to Botox, it is a “regular” in cosmetic hospitals. Botulinum toxin blocks the conduction between nerve endings and muscles through the nerve paralysis function, causing the muscles to relax, thus tightening the skin and eliminating lines, the same as facial muscle spasm is Botox. Botulinum toxin treatment for facial muscle spasm and the principle of wrinkle removal have similarities. Botox treatment for facial muscle spasm has a “shelf life” The director of the Department of Neurosurgery at Shanxi Provincial People’s Hospital, Ma Jihong, suggests that the effect of Botox treatment is temporary, generally an injection can last 3-6 months, but patients repeatedly injected: after repeated injections of Botox, the accumulation of toxins will lead to facial paralysis and facial deformation. This deformation cannot be recovered, and even if the patient chooses to have surgery in the future to cure the facial muscle spasm, the facial palsy will still exist. Etiology: A variety of causes can lead to facial spasm, such as: vascular compression of the facial nerve, recovery from facial neuritis (Bell’s palsy), occupying lesions in the pontocerebellar horn such as tumors and aneurysms, vascular malformations of the cerebellum, and facial hyperactivity. Of these, vascular compression is the most common factor. The only way to eradicate facial spasm – apparent microvascular decompression surgery There are many treatments for facial spasm, including medication, closure therapy, acupuncture and physiotherapy. Microvascular decompression surgery of the facial nerve is the preferred treatment, especially for patients who have been found to have a clear responsible vessel through MR examination, and the surgical risk is relatively small, the effect is very definite, and there are few complications. Under general anesthesia, an incision of about 5 cm is made behind the affected ear, and a skull window of about 2 cm is opened. The blood vessels compressing the facial nerve are cushioned from the facial nerve with a special material under the operating microscope, and the surgery causes almost no damage to the patient’s nerve and brain tissue. At present, microvascular decompression of the facial nerve is the best treatment method, and the spasm can be controlled immediately in more than 90% of patients, and gradually stopped in another part of patients in more than 1 week to 6 months.